Loss of the adaptor protein ShcA in endothelial cells protects against monocyte macrophage adhesion, LDL-oxydation, and atherosclerotic lesion formation
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2045-2322
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
-
Loss of the adaptor protein ShcA in endothelial cells protects against monocyte macrophage adhesion, LDL-oxydation, and atherosclerotic lesion formation ; volume:8 ; number:1 ; day:14 ; month:3 ; year:2018 ; pages:1-12 ; date:12.2018
Scientific reports ; 8, Heft 1 (14.3.2018), 1-12, 12.2018
- Creator
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Jaoude, Antoine Abou
- Contributor
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Badiqué, Lise
Mlih, Mohamed
Awan, Sara
Guo, Sunning
Lemle, Alexandre
Abboud, Clauda
Foppolo, Sophie
Host, Lionel
Terrand, Jérôme
Justiniano, Hélène
Herz, Joachim
Matz, Rachel L.
Boucher, Philippe
SpringerLink (Online service)
- DOI
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10.1038/s41598-018-22819-3
- URN
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urn:nbn:de:1111-2018051319322
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:50 AM CEST
Data provider
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Associated
- Jaoude, Antoine Abou
- Badiqué, Lise
- Mlih, Mohamed
- Awan, Sara
- Guo, Sunning
- Lemle, Alexandre
- Abboud, Clauda
- Foppolo, Sophie
- Host, Lionel
- Terrand, Jérôme
- Justiniano, Hélène
- Herz, Joachim
- Matz, Rachel L.
- Boucher, Philippe
- SpringerLink (Online service)
Other Objects (12)
Author Correction: Loss of the adaptor protein ShcA in endothelial cells protects against monocyte macrophage adhesion, LDL-oxydation, and atherosclerotic lesion formation
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Oxydation.
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Platelet activation and platelet-monocyte aggregate formation induced by the atherosclerotic plaque lipid lysophosphatidic acid
M2 macrophages exhibit higher sensitivity to oxLDL-induced lipotoxicity than other monocyte/macrophage subtypes
The correlation between lipoprotein(a) and coronary atherosclerotic lesion is stronger than LDL-C, when LDL-C is less than 104 mg/dL
Paraffin-Oxydation
Author Correction: Loss of the adaptor protein ShcA in endothelial cells protects against monocyte macrophage adhesion, LDL-oxydation, and atherosclerotic lesion formation
Ceruloplasmin and oxidized LDL colocalize in atherosclerotic lesions of hamster
Predictive value of LDL/HDL ratio in coronary atherosclerotic heart disease
Remnants, LDL, and the Quantification of Lipoprotein-Associated Risk in Atherosclerotic Cardiovascular Disease
Oxydation.
Oxydation.
Hydrogen-Peroxide Synthesis and LDL-Uptake Controls Immunosuppressive Properties in Monocyte-Derived Dendritic Cells
Molecular imaging of atherosclerotic plaque targeted to oxidized LDL receptor LOX-1 using magnetic resonance
Platelet activation and platelet-monocyte aggregate formation induced by the atherosclerotic plaque lipid lysophosphatidic acid
M2 macrophages exhibit higher sensitivity to oxLDL-induced lipotoxicity than other monocyte/macrophage subtypes
The correlation between lipoprotein(a) and coronary atherosclerotic lesion is stronger than LDL-C, when LDL-C is less than 104 mg/dL
Paraffin-Oxydation
Author Correction: Loss of the adaptor protein ShcA in endothelial cells protects against monocyte macrophage adhesion, LDL-oxydation, and atherosclerotic lesion formation
Ceruloplasmin and oxidized LDL colocalize in atherosclerotic lesions of hamster
Predictive value of LDL/HDL ratio in coronary atherosclerotic heart disease
Remnants, LDL, and the Quantification of Lipoprotein-Associated Risk in Atherosclerotic Cardiovascular Disease
Oxydation.
Oxydation.
Hydrogen-Peroxide Synthesis and LDL-Uptake Controls Immunosuppressive Properties in Monocyte-Derived Dendritic Cells
Molecular imaging of atherosclerotic plaque targeted to oxidized LDL receptor LOX-1 using magnetic resonance
Platelet activation and platelet-monocyte aggregate formation induced by the atherosclerotic plaque lipid lysophosphatidic acid
M2 macrophages exhibit higher sensitivity to oxLDL-induced lipotoxicity than other monocyte/macrophage subtypes
The correlation between lipoprotein(a) and coronary atherosclerotic lesion is stronger than LDL-C, when LDL-C is less than 104 mg/dL