Dual Inhibition of Bcl-2/Bcl-xL and XPO1 is synthetically lethal in glioblastoma model systems
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2045-2322
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Dual Inhibition of Bcl-2/Bcl-xL and XPO1 is synthetically lethal in glioblastoma model systems ; volume:8 ; number:1 ; day:18 ; month:10 ; year:2018 ; pages:1-11 ; date:12.2018
Scientific reports ; 8, Heft 1 (18.10.2018), 1-11, 12.2018
- Creator
-
Shang, Enyuan
- Contributor
-
Zhang, Yiru
Shu, Chang
Ishida, Chiaki Tsuge
Bianchetti, Elena
Westhoff, Mike-Andrew
Karpel-Massler, Georg
Siegelin, Markus D.
SpringerLink (Online service)
- DOI
-
10.1038/s41598-018-33784-2
- URN
-
urn:nbn:de:101:1-2018111716203541223699
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:52 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Shang, Enyuan
- Zhang, Yiru
- Shu, Chang
- Ishida, Chiaki Tsuge
- Bianchetti, Elena
- Westhoff, Mike-Andrew
- Karpel-Massler, Georg
- Siegelin, Markus D.
- SpringerLink (Online service)
Other Objects (12)
Inhibition of Bcl-2/Bcl-xL and c-MET causes synthetic lethality in model systems of glioblastoma
Epigenetic targeting of Mcl-1 Is synthetically lethal with Bcl-xL/Bcl-2 inhibition in model systems of glioblastoma
Bcl-XL but Not Bcl-2 is a potential target in medulloblastoma therapy
Regulation of Bcl-xL activity in apoptosis
Photodynamic therapy combined with Bcl-2/Bcl-xL inhibition increases the Noxa/Mcl-1 ratio independent of usp9X and synergistically enhances apoptosis in glioblastoma
Bcl-xl as the most promising Bcl-2 family member in targeted treatment of chondrosarcoma
BH4 domain peptides derived from Bcl-2/Bcl-XL as novel tools against acute pancreatitis
Bcl-xL inhibition enhances Dinaciclib-induced cell death in soft-tissue sarcomas
Transient apoptosis inhibition by Bcl-xL overexpression during hematopoietic stem cell transplantation
The downregulation of Mcl-1 via USP9X inhibition sensitizes solid tumors to Bcl-xl inhibition
Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms
BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival
Inhibition of Bcl-2/Bcl-xL and c-MET causes synthetic lethality in model systems of glioblastoma
Epigenetic targeting of Mcl-1 Is synthetically lethal with Bcl-xL/Bcl-2 inhibition in model systems of glioblastoma
Bcl-XL but Not Bcl-2 is a potential target in medulloblastoma therapy
Regulation of Bcl-xL activity in apoptosis
Photodynamic therapy combined with Bcl-2/Bcl-xL inhibition increases the Noxa/Mcl-1 ratio independent of usp9X and synergistically enhances apoptosis in glioblastoma
Bcl-xl as the most promising Bcl-2 family member in targeted treatment of chondrosarcoma
BH4 domain peptides derived from Bcl-2/Bcl-XL as novel tools against acute pancreatitis
Bcl-xL inhibition enhances Dinaciclib-induced cell death in soft-tissue sarcomas
Transient apoptosis inhibition by Bcl-xL overexpression during hematopoietic stem cell transplantation
The downregulation of Mcl-1 via USP9X inhibition sensitizes solid tumors to Bcl-xl inhibition
Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms
BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival
Inhibition of Bcl-2/Bcl-xL and c-MET causes synthetic lethality in model systems of glioblastoma
Epigenetic targeting of Mcl-1 Is synthetically lethal with Bcl-xL/Bcl-2 inhibition in model systems of glioblastoma
Bcl-XL but Not Bcl-2 is a potential target in medulloblastoma therapy
Regulation of Bcl-xL activity in apoptosis
Photodynamic therapy combined with Bcl-2/Bcl-xL inhibition increases the Noxa/Mcl-1 ratio independent of usp9X and synergistically enhances apoptosis in glioblastoma
Bcl-xl as the most promising Bcl-2 family member in targeted treatment of chondrosarcoma
BH4 domain peptides derived from Bcl-2/Bcl-XL as novel tools against acute pancreatitis
Bcl-xL inhibition enhances Dinaciclib-induced cell death in soft-tissue sarcomas
Transient apoptosis inhibition by Bcl-xL overexpression during hematopoietic stem cell transplantation
The downregulation of Mcl-1 via USP9X inhibition sensitizes solid tumors to Bcl-xl inhibition
Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms