Presenilin-1-Derived Circular RNAs : Neglected Epigenetic Regulators with Various Functions in Alzheimer’s Disease

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Bibliographic citation: In: 10.3390/biom13091401

Event: Veröffentlichung

(where): Aachen

(who): Universitätsbibliothek der RWTH Aachen

(when): 2023

Creator: Sanadgol, Nima
Amini, Javad
Beyer, Cordian
Zendedel, Adib

DOI: 10.18154/RWTH-CONV-252987

URN: urn:nbn:de:101:1-2406220145013.551248978784

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:54 AM CEST

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Sanadgol, Nima
Amini, Javad
Beyer, Cordian
Zendedel, Adib
Universitätsbibliothek der RWTH Aachen

Time of origin

2023

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Presenilin-1-Derived Circular RNAs : Neglected Epigenetic Regulators with Various Functions in Alzheimer’s Disease

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Other Objects (12)

Function and dysfunction of presenilin in Alzheimer’s disease

CSF Presenilin-1 complexes are increased in Alzheimer’s disease

Selective ferroptosis vulnerability due to familial Alzheimer’s disease presenilin mutations

Presenilin E318G variant and Alzheimer’s disease risk: the Cache County study

Cerebrospinal fluid Presenilin-1 increases at asymptomatic stage in genetically determined Alzheimer’s disease

Novel interaction between Alzheimer’s disease-related protein presenilin 1 and glutamate transporter 1

Pathological relevance of phosphorylation in the loop region of Presenilin 1 in Alzheimer’s disease

Generation and investigation of a faithful model for familial Alzheimer’s disease with presenilin mutations

Whole genome deconvolution unveils Alzheimer’s resilient epigenetic signature

The Role of Presenilin and its Interacting Proteins in the Biogenesis of Alzheimer’s Beta Amyloid

PLD3 epigenetic changes in the hippocampus of Alzheimer’s disease

MicroRNAs: pioneering regulators in Alzheimer’s disease pathogenesis, diagnosis, and therapy

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CSF Presenilin-1 complexes are increased in Alzheimer’s disease

Selective ferroptosis vulnerability due to familial Alzheimer’s disease presenilin mutations

Presenilin E318G variant and Alzheimer’s disease risk: the Cache County study

Cerebrospinal fluid Presenilin-1 increases at asymptomatic stage in genetically determined Alzheimer’s disease

Novel interaction between Alzheimer’s disease-related protein presenilin 1 and glutamate transporter 1

Pathological relevance of phosphorylation in the loop region of Presenilin 1 in Alzheimer’s disease

Generation and investigation of a faithful model for familial Alzheimer’s disease with presenilin mutations

Whole genome deconvolution unveils Alzheimer’s resilient epigenetic signature

The Role of Presenilin and its Interacting Proteins in the Biogenesis of Alzheimer’s Beta Amyloid

PLD3 epigenetic changes in the hippocampus of Alzheimer’s disease

MicroRNAs: pioneering regulators in Alzheimer’s disease pathogenesis, diagnosis, and therapy

Function and dysfunction of presenilin in Alzheimer’s disease

CSF Presenilin-1 complexes are increased in Alzheimer’s disease

Selective ferroptosis vulnerability due to familial Alzheimer’s disease presenilin mutations

Presenilin E318G variant and Alzheimer’s disease risk: the Cache County study

Cerebrospinal fluid Presenilin-1 increases at asymptomatic stage in genetically determined Alzheimer’s disease

Novel interaction between Alzheimer’s disease-related protein presenilin 1 and glutamate transporter 1

Pathological relevance of phosphorylation in the loop region of Presenilin 1 in Alzheimer’s disease

Generation and investigation of a faithful model for familial Alzheimer’s disease with presenilin mutations

Whole genome deconvolution unveils Alzheimer’s resilient epigenetic signature

The Role of Presenilin and its Interacting Proteins in the Biogenesis of Alzheimer’s Beta Amyloid

PLD3 epigenetic changes in the hippocampus of Alzheimer’s disease

MicroRNAs: pioneering regulators in Alzheimer’s disease pathogenesis, diagnosis, and therapy

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