- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1465-993X
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Abnormal spatial diffusion of Ca2+in F508del-CFTR airway epithelial cells ; volume:9 ; number:1 ; day:30 ; month:10 ; year:2008 ; pages:1-17 ; date:12.2008
Respiratory research ; 9, Heft 1 (30.10.2008), 1-17, 12.2008
- Creator
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Antigny, Fabrice
- Contributor
-
Norez, Caroline
Cantereau, Anne
Becq, Frédéric
Vandebrouck, Clarisse
SpringerLink (Online service)
- DOI
-
10.1186/1465-9921-9-70
- URN
-
urn:nbn:de:1111-201606039331
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:59 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Antigny, Fabrice
- Norez, Caroline
- Cantereau, Anne
- Becq, Frédéric
- Vandebrouck, Clarisse
- SpringerLink (Online service)
Other Objects (12)
The F508del-CFTR trafficking correctors elexacaftor and tezacaftor are CFTR-independent Ca2+-mobilizing agonists normalizing abnormal Ca2+ levels in human airway epithelial cells
Phosphodiesterase 8A Regulates CFTR Activity in Airway Epithelial Cells
Naringenin Regulates CFTR Activation and Expression in Airway Epithelial Cells
Modulation of cAMP metabolism for CFTR potentiation in human airway epithelial cells
Stepwise generation of CFTR-expressing airway epithelial cells from human pluripotent stem cells : application of a CFTR reporter cell line
Role of CFTR in epithelial physiology
Mutant CFTR Drives TWIST1 mediated epithelial–mesenchymal transition
Systematic optimization of prime editing for the efficient functional correction of CFTR F508del in human airway epithelial cells
CFTR dysfunction in smooth muscle drives TGFβ dependent airway hyperreactivity
CFTR expression decreases with age in several airway cell types
Increases in cytosolic Ca2+ induce dynamin- and calcineurin-dependent internalisation of CFTR
Control of epithelial Na+ channels by CFTR, receptors and kinases
The F508del-CFTR trafficking correctors elexacaftor and tezacaftor are CFTR-independent Ca2+-mobilizing agonists normalizing abnormal Ca2+ levels in human airway epithelial cells
Phosphodiesterase 8A Regulates CFTR Activity in Airway Epithelial Cells
Naringenin Regulates CFTR Activation and Expression in Airway Epithelial Cells
Modulation of cAMP metabolism for CFTR potentiation in human airway epithelial cells
Stepwise generation of CFTR-expressing airway epithelial cells from human pluripotent stem cells : application of a CFTR reporter cell line
Role of CFTR in epithelial physiology
Mutant CFTR Drives TWIST1 mediated epithelial–mesenchymal transition
Systematic optimization of prime editing for the efficient functional correction of CFTR F508del in human airway epithelial cells
CFTR dysfunction in smooth muscle drives TGFβ dependent airway hyperreactivity
CFTR expression decreases with age in several airway cell types
Increases in cytosolic Ca2+ induce dynamin- and calcineurin-dependent internalisation of CFTR
Control of epithelial Na+ channels by CFTR, receptors and kinases
The F508del-CFTR trafficking correctors elexacaftor and tezacaftor are CFTR-independent Ca2+-mobilizing agonists normalizing abnormal Ca2+ levels in human airway epithelial cells
Phosphodiesterase 8A Regulates CFTR Activity in Airway Epithelial Cells
Naringenin Regulates CFTR Activation and Expression in Airway Epithelial Cells
Modulation of cAMP metabolism for CFTR potentiation in human airway epithelial cells
Stepwise generation of CFTR-expressing airway epithelial cells from human pluripotent stem cells : application of a CFTR reporter cell line
Role of CFTR in epithelial physiology
Mutant CFTR Drives TWIST1 mediated epithelial–mesenchymal transition
Systematic optimization of prime editing for the efficient functional correction of CFTR F508del in human airway epithelial cells
CFTR dysfunction in smooth muscle drives TGFβ dependent airway hyperreactivity
CFTR expression decreases with age in several airway cell types
Increases in cytosolic Ca2+ induce dynamin- and calcineurin-dependent internalisation of CFTR