- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource, 1 online resource.
- Language
-
Englisch
- Bibliographic citation
-
Axonal Transport Defect in Gigaxonin Deficiency Rescued by Tubastatin A ; volume:20 ; number:4 ; day:2 ; month:6 ; year:2023 ; pages:1215-1228 ; date:7.2023
Neurotherapeutics ; 20, Heft 4 (2.6.2023), 1215-1228, 7.2023
- Creator
-
Nath, Banshi
Phaneuf, Daniel
Julien, Jean-Pierre
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1007/s13311-023-01393-1
- URN
-
urn:nbn:de:101:1-2023110610461012695393
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:56 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Nath, Banshi
- Phaneuf, Daniel
- Julien, Jean-Pierre
- SpringerLink (Online service)
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AFG3L2 deficiency impairs axonal transport of mitochondria dependent on ROS and tau levels
Fanca deficiency is associated with alterations in osteoclastogenesis that are rescued by TNFα
Mechanisms of Axonal Transport Defects in ALS
Messenger RNA transport on lysosomal vesicles maintains axonal mitochondrial homeostasis and prevents axonal degeneration
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LRRK2 deficiency induced mitochondrial Ca2+ efflux inhibition can be rescued by Na+/Ca2+/Li+ exchanger upregulation
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Rescued
Rescued!
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AFG3L2 deficiency impairs axonal transport of mitochondria dependent on ROS and tau levels
Fanca deficiency is associated with alterations in osteoclastogenesis that are rescued by TNFα
Mechanisms of Axonal Transport Defects in ALS
Messenger RNA transport on lysosomal vesicles maintains axonal mitochondrial homeostasis and prevents axonal degeneration
Christ rescued : = (Der gerettete Christus)
LRRK2 deficiency induced mitochondrial Ca2+ efflux inhibition can be rescued by Na+/Ca2+/Li+ exchanger upregulation
In vivo imaging of axonal degeneration and axonal transport in mouse models of multiple sclerosis
A new method for quantifying mitochondrial axonal transport