Regulatory chromatin rewiring promotes metabolic switching during adaptation to oncogenic receptor tyrosine kinase inhibition

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1476-5594

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Regulatory chromatin rewiring promotes metabolic switching during adaptation to oncogenic receptor tyrosine kinase inhibition ; volume:41 ; number:43 ; day:24 ; month:9 ; year:2022 ; pages:4808-4822 ; date:10.2022
Oncogene ; 41, Heft 43 (24.9.2022), 4808-4822, 10.2022

Creator: Ogden, Samuel
Carys, Kashmala
Ahmed, Ibrahim
Bruce, Jason
Sharrocks, Andrew D.

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41388-022-02465-w

URN: urn:nbn:de:101:1-2023010709135446829525

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:37 AM CEST

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Ogden, Samuel
Carys, Kashmala
Ahmed, Ibrahim
Bruce, Jason
Sharrocks, Andrew D.
SpringerLink (Online service)

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Regulatory chromatin rewiring promotes metabolic switching during adaptation to oncogenic receptor tyrosine kinase inhibition

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Other Objects (12)

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Chromatin de-condensation by switching substrate elasticity

Allosteric activation mechanism of a human oncogenic chromatin remodeler ALC1

Bruton's tyrosine kinase inhibition induces rewiring of proximal and distal B‐cell receptor signaling in mice

Discovery of oncogenic ROS1 missense mutations with sensitivity to tyrosine kinase inhibitors

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Oncogenic signaling-mediated regulation of chromatin during tumorigenesis

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Chromatin de-condensation by switching substrate elasticity

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Chromatin de-condensation by switching substrate elasticity

Allosteric activation mechanism of a human oncogenic chromatin remodeler ALC1

Bruton's tyrosine kinase inhibition induces rewiring of proximal and distal B‐cell receptor signaling in mice

Discovery of oncogenic ROS1 missense mutations with sensitivity to tyrosine kinase inhibitors

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Claudin-4 controls the receptor tyrosine kinase EphA2 pro-oncogenic switch through ?-catenin

Quantification and localization of oncogenic receptor tyrosine kinase variant transcripts using molecular inversion probes

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