A Fc-VEGF chimeric fusion enhances PD-L1 immunotherapy via inducing immune reprogramming and infiltration in the immunosuppressive tumor microenvironment
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1432-0851
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
A Fc-VEGF chimeric fusion enhances PD-L1 immunotherapy via inducing immune reprogramming and infiltration in the immunosuppressive tumor microenvironment ; day:27 ; month:7 ; year:2022 ; pages:1-19
Cancer immunology immunotherapy ; (27.7.2022), 1-19
- Urheber
-
Kuo, Cheng-Liang
Chou, Han-Yu
Lien, Hui-Wen
Yeh, Chia-An
Wang, Jing-Rong
Chen, Chung-Hsing
Fan, Chi-Chen
Hsu, Chih-Ping
Kao, Ting-Yu
Ko, Tai-Ming
Lee, Alan Yueh-Luen
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1007/s00262-022-03255-9
- URN
-
urn:nbn:de:101:1-2022100820124339394872
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:36 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Kuo, Cheng-Liang
- Chou, Han-Yu
- Lien, Hui-Wen
- Yeh, Chia-An
- Wang, Jing-Rong
- Chen, Chung-Hsing
- Fan, Chi-Chen
- Hsu, Chih-Ping
- Kao, Ting-Yu
- Ko, Tai-Ming
- Lee, Alan Yueh-Luen
- SpringerLink (Online service)
Ähnliche Objekte (12)
Reprogramming immunosuppressive myeloid cells facilitates immunotherapy for colorectal cancer
Senescence drives immunotherapy resistance by inducing an immunosuppressive tumor microenvironment
Chimeric antigen receptor T cells targeting PD-L1 suppress tumor growth
TIMELESS upregulates PD-L1 expression and exerts an immunosuppressive role in breast cancer
Zosuquidar Promotes Antitumor Immunity by Inducing Autophagic Degradation of PD‐L1
Tumor derived UBR5 promotes ovarian cancer growth and metastasis through inducing immunosuppressive macrophages
Cancer associated fibroblasts sculpt tumour microenvironment by recruiting monocytes and inducing immunosuppressive PD-1+ TAMs
Chimeric tumor modeling reveals role of partial PDL1 expression in resistance to virally induced immunotherapy
An Optogenetic‐Controlled Cell Reprogramming System for Driving Cell Fate and Light‐Responsive Chimeric Mice
Elevation of spermine remodels immunosuppressive microenvironment through driving the modification of PD-L1 in hepatocellular carcinoma
Manipulating the Crosstalk between Cancer and Immunosuppressive Cells with Phototherapeutic Gold‐Nanohut for Reprogramming Tumor Microenvironment
Loss of LECT2 promotes ovarian cancer progression by inducing cancer invasiveness and facilitating an immunosuppressive environment
Reprogramming immunosuppressive myeloid cells facilitates immunotherapy for colorectal cancer
Senescence drives immunotherapy resistance by inducing an immunosuppressive tumor microenvironment
Chimeric antigen receptor T cells targeting PD-L1 suppress tumor growth
TIMELESS upregulates PD-L1 expression and exerts an immunosuppressive role in breast cancer
Zosuquidar Promotes Antitumor Immunity by Inducing Autophagic Degradation of PD‐L1
Tumor derived UBR5 promotes ovarian cancer growth and metastasis through inducing immunosuppressive macrophages
Cancer associated fibroblasts sculpt tumour microenvironment by recruiting monocytes and inducing immunosuppressive PD-1+ TAMs
Chimeric tumor modeling reveals role of partial PDL1 expression in resistance to virally induced immunotherapy
An Optogenetic‐Controlled Cell Reprogramming System for Driving Cell Fate and Light‐Responsive Chimeric Mice
Elevation of spermine remodels immunosuppressive microenvironment through driving the modification of PD-L1 in hepatocellular carcinoma
Manipulating the Crosstalk between Cancer and Immunosuppressive Cells with Phototherapeutic Gold‐Nanohut for Reprogramming Tumor Microenvironment
Loss of LECT2 promotes ovarian cancer progression by inducing cancer invasiveness and facilitating an immunosuppressive environment
Reprogramming immunosuppressive myeloid cells facilitates immunotherapy for colorectal cancer
Senescence drives immunotherapy resistance by inducing an immunosuppressive tumor microenvironment
Chimeric antigen receptor T cells targeting PD-L1 suppress tumor growth
TIMELESS upregulates PD-L1 expression and exerts an immunosuppressive role in breast cancer
Zosuquidar Promotes Antitumor Immunity by Inducing Autophagic Degradation of PD‐L1
Tumor derived UBR5 promotes ovarian cancer growth and metastasis through inducing immunosuppressive macrophages
Cancer associated fibroblasts sculpt tumour microenvironment by recruiting monocytes and inducing immunosuppressive PD-1+ TAMs
Chimeric tumor modeling reveals role of partial PDL1 expression in resistance to virally induced immunotherapy
An Optogenetic‐Controlled Cell Reprogramming System for Driving Cell Fate and Light‐Responsive Chimeric Mice
Elevation of spermine remodels immunosuppressive microenvironment through driving the modification of PD-L1 in hepatocellular carcinoma
Manipulating the Crosstalk between Cancer and Immunosuppressive Cells with Phototherapeutic Gold‐Nanohut for Reprogramming Tumor Microenvironment