- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
1 Online-Ressource.
- Sprache
-
Englisch
- Erschienen in
-
Lipoprotein(a): from Causality to Treatment ; volume:26 ; number:3 ; day:22 ; month:1 ; year:2024 ; pages:75-82 ; date:3.2024
Current atherosclerosis reports ; 26, Heft 3 (22.1.2024), 75-82, 3.2024
- Klassifikation
-
Medizin, Gesundheit
- Urheber
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1007/s11883-024-01187-6
- URN
-
urn:nbn:de:101:1-2404291333441.119297126172
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 11:00 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Kronenberg, Florian
- SpringerLink (Online service)
Ähnliche Objekte (12)
Human Genetics and the Causal Role of Lipoprotein(a) for Various Diseases
Prediction of cardiovascular risk by Lp(a) concentrations or genetic variants within the LPA gene region
Lipoprotein(a) plasma levels are not associated with incident microvascular complications in type 2 diabetes mellitus
Lipoprotein(a)
Frequent LPA KIV-2 variants lower lipoprotein(a) concentrations and protect against coronary artery disease
Lipoprotein(a) and incident type-2 diabetes: results from the prospective Bruneck study and a meta-analysis of published literature
Genomweite Assoziationsstudien zu Adipositas und was wir daraus lernen
An optimized procedure for the design and evaluation of Ecotilling assays
The effect of LPA Thr3888Pro on lipoprotein(a) and coronary artery disease is modified by the LPA KIV-2 variant 4925G>A
Lipoprotein(a) in nephrological patients
Effect of diet-induced weight loss on lipoprotein(a) levels in obese individuals with and without type 2 diabetes
Supporting the evidence for human trait-associated genetic variants by computational biology methods and multi-level data integration.
Human Genetics and the Causal Role of Lipoprotein(a) for Various Diseases
Prediction of cardiovascular risk by Lp(a) concentrations or genetic variants within the LPA gene region
Lipoprotein(a) plasma levels are not associated with incident microvascular complications in type 2 diabetes mellitus
Lipoprotein(a)
Frequent LPA KIV-2 variants lower lipoprotein(a) concentrations and protect against coronary artery disease
Lipoprotein(a) and incident type-2 diabetes: results from the prospective Bruneck study and a meta-analysis of published literature
Genomweite Assoziationsstudien zu Adipositas und was wir daraus lernen
An optimized procedure for the design and evaluation of Ecotilling assays
The effect of LPA Thr3888Pro on lipoprotein(a) and coronary artery disease is modified by the LPA KIV-2 variant 4925G>A
Lipoprotein(a) in nephrological patients
Effect of diet-induced weight loss on lipoprotein(a) levels in obese individuals with and without type 2 diabetes
Supporting the evidence for human trait-associated genetic variants by computational biology methods and multi-level data integration.
Human Genetics and the Causal Role of Lipoprotein(a) for Various Diseases
Prediction of cardiovascular risk by Lp(a) concentrations or genetic variants within the LPA gene region
Lipoprotein(a) plasma levels are not associated with incident microvascular complications in type 2 diabetes mellitus
Lipoprotein(a)
Frequent LPA KIV-2 variants lower lipoprotein(a) concentrations and protect against coronary artery disease
Lipoprotein(a) and incident type-2 diabetes: results from the prospective Bruneck study and a meta-analysis of published literature
Genomweite Assoziationsstudien zu Adipositas und was wir daraus lernen
An optimized procedure for the design and evaluation of Ecotilling assays
The effect of LPA Thr3888Pro on lipoprotein(a) and coronary artery disease is modified by the LPA KIV-2 variant 4925G>A
Lipoprotein(a) in nephrological patients
Effect of diet-induced weight loss on lipoprotein(a) levels in obese individuals with and without type 2 diabetes