IL-2 augments the sorafenib-induced apoptosis in liver cancer by promoting mitochondrial fission and activating the JNK/TAZ pathway
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1475-2867
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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IL-2 augments the sorafenib-induced apoptosis in liver cancer by promoting mitochondrial fission and activating the JNK/TAZ pathway ; volume:18 ; number:1 ; day:9 ; month:11 ; year:2018 ; pages:1-16 ; date:12.2018
Cancer cell international ; 18, Heft 1 (9.11.2018), 1-16, 12.2018
- Creator
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Ding, Xiaoyan
Sun, Wei
Chen, Jinglong
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s12935-018-0671-3
- URN
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urn:nbn:de:101:1-2018122723593131136611
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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2025-08-14T10:48:50+0200
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Ding, Xiaoyan
- Sun, Wei
- Chen, Jinglong
- SpringerLink (Online service)
Other Objects (12)
Retraction Note to: IL‑2 augments the sorafenib‑induced apoptosis in liver cancer by promoting mitochondrial fission and activating the JNK/TAZ pathway
TAZ inhibition promotes IL-2-induced apoptosis of hepatocellular carcinoma cells by activating the JNK/F-actin/mitochondrial fission pathway
Mst1 overexpression combined with Yap knockdown augments thyroid carcinoma apoptosis via promoting MIEF1-related mitochondrial fission and activating the JNK pathway
Retraction Note: Mst1 overexpression combined with Yap knockdown augments thyroid carcinoma apoptosis via promoting MIEF1-related mitochondrial fission and activating the JNK pathway
Mst1 regulates post-infarction cardiac injury through the JNK-Drp1-mitochondrial fission pathway
YAP/TAZ and ATF4 drive resistance to Sorafenib in hepatocellular carcinoma by preventing ferroptosis
taz münster / nrw taz Münster
TAZ
"TAZ"
Taz
TAZ
"TAZ"
Retraction Note to: IL‑2 augments the sorafenib‑induced apoptosis in liver cancer by promoting mitochondrial fission and activating the JNK/TAZ pathway
TAZ inhibition promotes IL-2-induced apoptosis of hepatocellular carcinoma cells by activating the JNK/F-actin/mitochondrial fission pathway
Mst1 overexpression combined with Yap knockdown augments thyroid carcinoma apoptosis via promoting MIEF1-related mitochondrial fission and activating the JNK pathway
Retraction Note: Mst1 overexpression combined with Yap knockdown augments thyroid carcinoma apoptosis via promoting MIEF1-related mitochondrial fission and activating the JNK pathway
Mst1 regulates post-infarction cardiac injury through the JNK-Drp1-mitochondrial fission pathway
YAP/TAZ and ATF4 drive resistance to Sorafenib in hepatocellular carcinoma by preventing ferroptosis
taz münster / nrw taz Münster
TAZ
"TAZ"
Taz
TAZ
"TAZ"
Retraction Note to: IL‑2 augments the sorafenib‑induced apoptosis in liver cancer by promoting mitochondrial fission and activating the JNK/TAZ pathway
TAZ inhibition promotes IL-2-induced apoptosis of hepatocellular carcinoma cells by activating the JNK/F-actin/mitochondrial fission pathway
Mst1 overexpression combined with Yap knockdown augments thyroid carcinoma apoptosis via promoting MIEF1-related mitochondrial fission and activating the JNK pathway
Retraction Note: Mst1 overexpression combined with Yap knockdown augments thyroid carcinoma apoptosis via promoting MIEF1-related mitochondrial fission and activating the JNK pathway
Mst1 regulates post-infarction cardiac injury through the JNK-Drp1-mitochondrial fission pathway
YAP/TAZ and ATF4 drive resistance to Sorafenib in hepatocellular carcinoma by preventing ferroptosis
taz münster / nrw taz Münster
TAZ
"TAZ"
Taz
TAZ