Glomerular spatial transcriptomics of IgA nephropathy according to the presence of mesangial proliferation
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Glomerular spatial transcriptomics of IgA nephropathy according to the presence of mesangial proliferation ; volume:14 ; number:1 ; day:26 ; month:1 ; year:2024 ; pages:1-12 ; date:12.2024
Scientific reports ; 14, Heft 1 (26.1.2024), 1-12, 12.2024
- Creator
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Park, Sehoon
Kang, Minji
Kim, Yong Chul
Kim, Dong Ki
Oh, Kook-Hwan
Joo, Kwon Wook
Kim, Yon Su
Kim, Hyun Je
Moon, Kyung Chul
Lee, Hajeong
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41598-024-52581-8
- URN
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urn:nbn:de:101:1-2024040810071472625252
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:47 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Park, Sehoon
- Kang, Minji
- Kim, Yong Chul
- Kim, Dong Ki
- Oh, Kook-Hwan
- Joo, Kwon Wook
- Kim, Yon Su
- Kim, Hyun Je
- Moon, Kyung Chul
- Lee, Hajeong
- SpringerLink (Online service)
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Glomerular tip adhesions predict the progression of IgA nephropathy
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Comparative proteomic analysis of glomerular proteins in IgA nephropathy and IgA vasculitis with nephritis
Mesangial cells from patients with IgA nephropathy have increased susceptibility to galactose-deficient IgA1
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Salvianolic Acid B Inhibits Oxidative Stress in Glomerular Mesangial Cells Alleviating Diabetic Nephropathy by Regulating SIRT3/FOXO1 Signaling
Low birth weight associates with glomerular area in young male IgA nephropathy patients
Glomerular C4d in Post-Transplant IgA Nephropathy is associated with decreased allograft survival
Intestinal permeability in patients with IgA nephropathy and other glomerular diseases: an observational study
Intestinal permeability in patients with IgA nephropathy and other glomerular diseases: an observational study
Histological reappraisal of IgA nephropathy: the role of glomerular pattern of injury and mesangial complement deposition
Pathophysiology of mesangial expansion in diabetic nephropathy: mesangial structure, glomerular biomechanics, and biochemical signaling and regulation
Glomerular tip adhesions predict the progression of IgA nephropathy
Role of Human Mesangial-Tubular Crosstalk in Secretory IgA-Induced IgA Nephropathy
Comparative proteomic analysis of glomerular proteins in IgA nephropathy and IgA vasculitis with nephritis
Mesangial cells from patients with IgA nephropathy have increased susceptibility to galactose-deficient IgA1
Tonsillectomy reduces recurrence of IgA nephropathy in mesangial hypercellularity type categorized by the Oxford classification
Salvianolic Acid B Inhibits Oxidative Stress in Glomerular Mesangial Cells Alleviating Diabetic Nephropathy by Regulating SIRT3/FOXO1 Signaling
Low birth weight associates with glomerular area in young male IgA nephropathy patients
Glomerular C4d in Post-Transplant IgA Nephropathy is associated with decreased allograft survival
Intestinal permeability in patients with IgA nephropathy and other glomerular diseases: an observational study