Mechanisms of Caloric Restriction-Mediated Stress-Resistance in Acute Kidney Injury

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Caloric restriction (CR)-mediated organ protection has been shown to be extremely efficient in rodent models of acute kidney injury (AKI). Limited understanding of the underlying mechanisms paired with a risk of malnourishment and feasibility problems has hindered the translation of this immense potential to the patient setting. In this mini-review, the current mechanistic concepts of CR-mediated stress-resistance as potential key targets for renal protection in AKI will be highlighted.

Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Bibliographic citation: Mechanisms of Caloric Restriction-Mediated Stress-Resistance in Acute Kidney Injury ; volume:146 ; number:3 ; year:2022 ; pages:234-238 ; extent:5
Nephron ; 146, Heft 3 (2022), 234-238 (gesamt 5)

Creator: Koehler, Felix C.
Späth, Martin R.
Hoyer-Allo, K. Johanna R.
Müller, Roman-Ulrich

DOI: 10.1159/000517733

URN: urn:nbn:de:101:1-2022051900414878320372

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:39 AM CEST

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Koehler, Felix C.
Späth, Martin R.
Hoyer-Allo, K. Johanna R.
Müller, Roman-Ulrich

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Mechanisms of Caloric Restriction-Mediated Stress-Resistance in Acute Kidney Injury

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Other Objects (12)

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Correction to: Caloric restriction induces anabolic resistance to resistance exercise

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Status of selected nutrients in obese dogs undergoing caloric restriction

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Caloric restriction in female reproduction: is it beneficial or detrimental?

Correction to: Caloric restriction induces anabolic resistance to resistance exercise

Caloric restriction leads to druggable LSD1-dependent cancer stem cells expansion

Genome-wide methylation is modified by caloric restriction in Daphnia magna

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