MiR-193a-3p is an Important Tumour Suppressor in Lung Cancer and Directly Targets KRAS
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Bibliographic citation
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MiR-193a-3p is an Important Tumour Suppressor in Lung Cancer and Directly Targets KRAS ; volume:44 ; number:4 ; year:2017 ; pages:1311-1324
Cellular physiology and biochemistry ; 44, Heft 4 (2017), 1311-1324
- Creator
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Fan, Qian
Hu, Xiuting
Zhang, Haiyang
Wang, Shengguang
Zhang, Huilai
You, Chaoying
Zhang, Chen-Yu
Liang, Hongwei
Chen, Xi
Ba, Yi
- DOI
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10.1159/000485491
- URN
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urn:nbn:de:101:1-2017122213536
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:37 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Fan, Qian
- Hu, Xiuting
- Zhang, Haiyang
- Wang, Shengguang
- Zhang, Huilai
- You, Chaoying
- Zhang, Chen-Yu
- Liang, Hongwei
- Chen, Xi
- Ba, Yi
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microRNA-193a-3p is specifically down-regulated and acts as a tumor suppressor in BRAF-mutated colorectal cancer
Correction: miR-148b-3p functions as a tumor suppressor in GISTs by directly targeting KIT
193a
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MiR-193a-3p and miR-193a-5p suppress the metastasis of human osteosarcoma cells by down-regulating Rab27B and SRR, respectively
Oncogenic KRAS Drives Metabolic Vulnerabilities by Directly Regulating Metabolic Enzymes in Cancer
Human tumor suppressor PDCD4 directly interacts with ribosomes to repress translation
Kölner Landstraße 193a
miR-148b-3p functions as a tumor suppressor in GISTs by directly targeting KIT
microRNA-193a-3p is specifically down-regulated and acts as a tumor suppressor in BRAF-mutated colorectal cancer
Correction: miR-148b-3p functions as a tumor suppressor in GISTs by directly targeting KIT
193a
Höherweg 193a
Grabstein 193A
Grabstein 193A
Grabstein 193A
MiR-193a-3p and miR-193a-5p suppress the metastasis of human osteosarcoma cells by down-regulating Rab27B and SRR, respectively
Oncogenic KRAS Drives Metabolic Vulnerabilities by Directly Regulating Metabolic Enzymes in Cancer
Human tumor suppressor PDCD4 directly interacts with ribosomes to repress translation