- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource, 24 Seiten
- Language
-
Englisch
- Bibliographic citation
-
In: Cells(13), H. 9, S.1-24 - ISSN 2073-4409
In: © 2024 by the authors., Basel
- Event
-
Veröffentlichung
- (where)
-
Jena
- (who)
-
Friedrich-Schiller-Universität Jena
- (when)
-
2024
- Creator
- Contributor
-
Fabrizio, Paola
- URN
-
urn:nbn:de:gbv:27-dbt-60851-1
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:55 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Ohse, Verena Alexia
- Klotz, Lars-Oliver
- Priebs, Josephine
- Fabrizio, Paola
- Friedrich-Schiller-Universität Jena
Time of origin
- 2024
Other Objects (12)
Natural products against neurodegenerative diseases: effects in the model organism Caenorhabditis elegans
C. elegans is not a robust model organism for the magnetic sense
Caenorhabditis elegans as a model organism for RYR1 variants and muscle ageing
Control of glutamate homeostasis in the Gram-positive model organism Bacillus subtilis
Reactive oxygen species-mediated signaling promotes lifespan of the model organism Caenorhabditis elegans
Characterization of chaperone signatures and neurodegenerative diseases using the model organism C. elegans
Genetic variation in neurodegenerative diseases and its accessibility in the model organism Caenorhabditis elegans
Advancing insights into microgravity induced muscle changes using Caenorhabditis elegans as a model organism
A fast and reliable method for monitoring genomic instability in the model organism Caenorhabditis elegans
Mitochondrial copper homeostasis in mammalian cells
C. elegans as model organism for the identification of new components of the TOR signaling pathway
Unique underlying principles shaping copper homeostasis networks
Natural products against neurodegenerative diseases: effects in the model organism Caenorhabditis elegans
C. elegans is not a robust model organism for the magnetic sense
Caenorhabditis elegans as a model organism for RYR1 variants and muscle ageing
Control of glutamate homeostasis in the Gram-positive model organism Bacillus subtilis
Reactive oxygen species-mediated signaling promotes lifespan of the model organism Caenorhabditis elegans
Characterization of chaperone signatures and neurodegenerative diseases using the model organism C. elegans
Genetic variation in neurodegenerative diseases and its accessibility in the model organism Caenorhabditis elegans
Advancing insights into microgravity induced muscle changes using Caenorhabditis elegans as a model organism
A fast and reliable method for monitoring genomic instability in the model organism Caenorhabditis elegans
Mitochondrial copper homeostasis in mammalian cells
C. elegans as model organism for the identification of new components of the TOR signaling pathway
Unique underlying principles shaping copper homeostasis networks
Natural products against neurodegenerative diseases: effects in the model organism Caenorhabditis elegans
C. elegans is not a robust model organism for the magnetic sense
Caenorhabditis elegans as a model organism for RYR1 variants and muscle ageing
Control of glutamate homeostasis in the Gram-positive model organism Bacillus subtilis
Reactive oxygen species-mediated signaling promotes lifespan of the model organism Caenorhabditis elegans
Characterization of chaperone signatures and neurodegenerative diseases using the model organism C. elegans
Genetic variation in neurodegenerative diseases and its accessibility in the model organism Caenorhabditis elegans
Advancing insights into microgravity induced muscle changes using Caenorhabditis elegans as a model organism
A fast and reliable method for monitoring genomic instability in the model organism Caenorhabditis elegans
Mitochondrial copper homeostasis in mammalian cells
C. elegans as model organism for the identification of new components of the TOR signaling pathway