Inhibiting WNT secretion reduces high bone mass caused by Sost loss-of-function or gain-of-function mutations in Lrp5
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
1 Online-Ressource.
- Language
-
Englisch
- Bibliographic citation
-
Inhibiting WNT secretion reduces high bone mass caused by Sost loss-of-function or gain-of-function mutations in Lrp5 ; volume:11 ; number:1 ; day:24 ; month:8 ; year:2023 ; pages:1-12 ; date:12.2023
Bone research ; 11, Heft 1 (24.8.2023), 1-12, 12.2023
- Creator
-
Diegel, Cassandra R.
Kramer, Ina
Moes, Charles
Foxa, Gabrielle E.
McDonald, Mitchell J.
Madaj, Zachary B.
Guth, Sabine
Liu, Jun
Harris, Jennifer L.
Kneissel, Michaela
Williams, Bart O.
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1038/s41413-023-00278-5
- URN
-
urn:nbn:de:101:1-2024020514540002721328
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:34 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Diegel, Cassandra R.
- Kramer, Ina
- Moes, Charles
- Foxa, Gabrielle E.
- McDonald, Mitchell J.
- Madaj, Zachary B.
- Guth, Sabine
- Liu, Jun
- Harris, Jennifer L.
- Kneissel, Michaela
- Williams, Bart O.
- SpringerLink (Online service)
Other Objects (12)
Characterization of a new mouse p53 variant: loss-of-function and gain-of-function
Loss-of-function, gain-of-function and dominant-negative mutations have profoundly different effects on protein structure
JAKs and STATs from a clinical perspective: loss-of-function mutations, gain-of-function mutations, and their multidimensional consequences
Egfr-signaling in Oryzias latipes - Characterization of inducible oncogene gain-of-function and receptor loss-of-function Medaka lines
Gain-of-function and loss-of-function GABRB3 variants lead to distinct clinical phenotypes in patients with developmental and epileptic encephalopathies
Gain-of-Function Research: Ethical Analysis
Mutant p53 gain-of-function stimulates canonical Wnt signaling via PI3K/AKT pathway in colon cancer
MDA5 gain-of-function associated with a Glu794del mutation
PITX2 gain-of-function induced defects in mouse forelimb development
The curious case of Gαs gain-of-function in neoplasia
A synergic effect between CYP2C19*2, CYP2C19*3 loss-of-function and CYP2C19*17 gain-of-function alleles is associated with Clopidogrel resistance among Moroccan Acute Coronary Syndromes patients
Loss-of-function uORF mutations in human malignancies
Characterization of a new mouse p53 variant: loss-of-function and gain-of-function
Loss-of-function, gain-of-function and dominant-negative mutations have profoundly different effects on protein structure
JAKs and STATs from a clinical perspective: loss-of-function mutations, gain-of-function mutations, and their multidimensional consequences
Egfr-signaling in Oryzias latipes - Characterization of inducible oncogene gain-of-function and receptor loss-of-function Medaka lines
Gain-of-function and loss-of-function GABRB3 variants lead to distinct clinical phenotypes in patients with developmental and epileptic encephalopathies
Gain-of-Function Research: Ethical Analysis
Mutant p53 gain-of-function stimulates canonical Wnt signaling via PI3K/AKT pathway in colon cancer
MDA5 gain-of-function associated with a Glu794del mutation
PITX2 gain-of-function induced defects in mouse forelimb development
The curious case of Gαs gain-of-function in neoplasia
A synergic effect between CYP2C19*2, CYP2C19*3 loss-of-function and CYP2C19*17 gain-of-function alleles is associated with Clopidogrel resistance among Moroccan Acute Coronary Syndromes patients
Loss-of-function uORF mutations in human malignancies
Characterization of a new mouse p53 variant: loss-of-function and gain-of-function
Loss-of-function, gain-of-function and dominant-negative mutations have profoundly different effects on protein structure
JAKs and STATs from a clinical perspective: loss-of-function mutations, gain-of-function mutations, and their multidimensional consequences
Egfr-signaling in Oryzias latipes - Characterization of inducible oncogene gain-of-function and receptor loss-of-function Medaka lines
Gain-of-function and loss-of-function GABRB3 variants lead to distinct clinical phenotypes in patients with developmental and epileptic encephalopathies
Gain-of-Function Research: Ethical Analysis
Mutant p53 gain-of-function stimulates canonical Wnt signaling via PI3K/AKT pathway in colon cancer
MDA5 gain-of-function associated with a Glu794del mutation
PITX2 gain-of-function induced defects in mouse forelimb development
The curious case of Gαs gain-of-function in neoplasia
A synergic effect between CYP2C19*2, CYP2C19*3 loss-of-function and CYP2C19*17 gain-of-function alleles is associated with Clopidogrel resistance among Moroccan Acute Coronary Syndromes patients