Elimination of the four extracellular matrix molecules tenascin-C, tenascin-R, brevican and neurocan alters the ratio of excitatory and inhibitory synapses
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Bibliographic citation
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In: 10.1038/s41598-019-50404-9
- Event
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Veröffentlichung
- (where)
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Aachen
- (who)
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Universitätsbibliothek der RWTH Aachen
- (when)
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2019
- Creator
- DOI
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10.18154/RWTH-2020-01851
- URN
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urn:nbn:de:101:1-2021101806441939076927
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
- 15.08.2025, 7:35 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Gottschling, Christine
- Wegrzyn, David
- Denecke, Bernd
- Faissner, Andreas
- Universitätsbibliothek der RWTH Aachen
Time of origin
- 2019
Other Objects (12)
Elimination of the four extracellular matrix molecules tenascin-C, tenascin-R, brevican and neurocan alters the ratio of excitatory and inhibitory synapses
Identification, cloning and characterization of the novel tenascin family member tenascin-N and conditional gene targeting of tenascin-R
Identification, cloning and characterization of the novel tenascin family member tenascin-N and conditional gene targeting of tenascin-R
Advances in tenascin-C biology
Brevican, Neurocan, Tenascin-C, and Tenascin-R act as important regulators of the interplay between perineuronal nets, synaptic integrity, inhibitory interneurons, and Otx2
Tenascin-W is a better cancer biomarker than tenascin-C for most human solid tumors
INEX – A binary neuronal model with inhibitory and excitatory synapses
Raised circulating tenascin-C in rheumatoid arthritis
Tenascin-C modulates alveolarization in bronchopulmonary dysplasia
The extracellular matrix molecules tenascin-C, tenascin-R, brevican and neurocan regulate perineural nets, synaptogenesis and neuronal network activity
Expression von Tenascin beim Magenkarzinom : eine immunhistochemische Untersuchung
Signaltransduktion des Tenascin-C beim Neuritenwachstum von Hippokampusneuronen
Elimination of the four extracellular matrix molecules tenascin-C, tenascin-R, brevican and neurocan alters the ratio of excitatory and inhibitory synapses
Identification, cloning and characterization of the novel tenascin family member tenascin-N and conditional gene targeting of tenascin-R
Identification, cloning and characterization of the novel tenascin family member tenascin-N and conditional gene targeting of tenascin-R
Advances in tenascin-C biology
Brevican, Neurocan, Tenascin-C, and Tenascin-R act as important regulators of the interplay between perineuronal nets, synaptic integrity, inhibitory interneurons, and Otx2
Tenascin-W is a better cancer biomarker than tenascin-C for most human solid tumors
INEX – A binary neuronal model with inhibitory and excitatory synapses
Raised circulating tenascin-C in rheumatoid arthritis
Tenascin-C modulates alveolarization in bronchopulmonary dysplasia
The extracellular matrix molecules tenascin-C, tenascin-R, brevican and neurocan regulate perineural nets, synaptogenesis and neuronal network activity
Expression von Tenascin beim Magenkarzinom : eine immunhistochemische Untersuchung
Signaltransduktion des Tenascin-C beim Neuritenwachstum von Hippokampusneuronen
Elimination of the four extracellular matrix molecules tenascin-C, tenascin-R, brevican and neurocan alters the ratio of excitatory and inhibitory synapses
Identification, cloning and characterization of the novel tenascin family member tenascin-N and conditional gene targeting of tenascin-R
Identification, cloning and characterization of the novel tenascin family member tenascin-N and conditional gene targeting of tenascin-R
Advances in tenascin-C biology
Brevican, Neurocan, Tenascin-C, and Tenascin-R act as important regulators of the interplay between perineuronal nets, synaptic integrity, inhibitory interneurons, and Otx2
Tenascin-W is a better cancer biomarker than tenascin-C for most human solid tumors
INEX – A binary neuronal model with inhibitory and excitatory synapses
Raised circulating tenascin-C in rheumatoid arthritis
Tenascin-C modulates alveolarization in bronchopulmonary dysplasia
The extracellular matrix molecules tenascin-C, tenascin-R, brevican and neurocan regulate perineural nets, synaptogenesis and neuronal network activity
Expression von Tenascin beim Magenkarzinom : eine immunhistochemische Untersuchung