Elimination of the four extracellular matrix molecules tenascin-C, tenascin-R, brevican and neurocan alters the ratio of excitatory and inhibitory synapses
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Erschienen in
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In: Scientific reports, 9, Artikel 13939, S. 13939-1-13939-17
- Ereignis
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Veröffentlichung
- (wo)
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Bochum
- (wer)
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Ruhr-Universität Bochum
- (wann)
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2019
- Urheber
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Gottschling, Christine
Wegrzyn, David
Denecke, Bernd
Faissner, Andreas
- URN
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urn:nbn:de:hbz:294-73180
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
25.03.2025, 13:49 MEZ
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Beteiligte
- Gottschling, Christine
- Wegrzyn, David
- Denecke, Bernd
- Faissner, Andreas
- Ruhr-Universität Bochum
Entstanden
- 2019
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Elimination of the four extracellular matrix molecules tenascin-C, tenascin-R, brevican and neurocan alters the ratio of excitatory and inhibitory synapses
The extracellular matrix proteins tenascin-C and tenascin-R retard oligodendrocyte precursor maturation and myelin regeneration in a cuprizone-induced long-term demyelination animal model
Brevican, Neurocan, Tenascin-C, and Tenascin-R act as important regulators of the interplay between perineuronal nets, synaptic integrity, inhibitory interneurons, and Otx2
Substrate-bound and soluble domains of tenascin-C regulate differentiation, proliferation and migration of neural stem and progenitor cells
Cell tracking in vitro reveals that the extracellular matrix glycoprotein Tenascin-C
Role of the extracellular matrix glycoprotein Tenascin-c in brain ischemia and ist regulation by Erythropoietin
Modulation of GluN3B-containing excitatory glycine receptors by zinc
Low-density lipoprotein receptor-related protein-1 (LRP1) in the glial lineage modulates neuronal excitability
The guanine nucleotide exchange factor Vav3 intervenes in the migration pathway of oligodendrocyte precursor cells on tenascin-C
Cell tracking in vitro reveals that the extracellular matrix glycoprotein Tenascin-C modulates cell cycle length and differentiation in neural stem/progenitor cells of the developing mouse spinal cord
Loss of the extracellular matrix molecule tenascin-C leads to absence of reactive gliosis and promotes anti-inflammatory cytokine expression in an autoimmune glaucoma mouse model
Identification, cloning and characterization of the novel tenascin family member tenascin-N and conditional gene targeting of tenascin-R
Elimination of the four extracellular matrix molecules tenascin-C, tenascin-R, brevican and neurocan alters the ratio of excitatory and inhibitory synapses
The extracellular matrix proteins tenascin-C and tenascin-R retard oligodendrocyte precursor maturation and myelin regeneration in a cuprizone-induced long-term demyelination animal model
Brevican, Neurocan, Tenascin-C, and Tenascin-R act as important regulators of the interplay between perineuronal nets, synaptic integrity, inhibitory interneurons, and Otx2
Substrate-bound and soluble domains of tenascin-C regulate differentiation, proliferation and migration of neural stem and progenitor cells
Cell tracking in vitro reveals that the extracellular matrix glycoprotein Tenascin-C
Role of the extracellular matrix glycoprotein Tenascin-c in brain ischemia and ist regulation by Erythropoietin
Modulation of GluN3B-containing excitatory glycine receptors by zinc
Low-density lipoprotein receptor-related protein-1 (LRP1) in the glial lineage modulates neuronal excitability
The guanine nucleotide exchange factor Vav3 intervenes in the migration pathway of oligodendrocyte precursor cells on tenascin-C
Cell tracking in vitro reveals that the extracellular matrix glycoprotein Tenascin-C modulates cell cycle length and differentiation in neural stem/progenitor cells of the developing mouse spinal cord
Loss of the extracellular matrix molecule tenascin-C leads to absence of reactive gliosis and promotes anti-inflammatory cytokine expression in an autoimmune glaucoma mouse model
Identification, cloning and characterization of the novel tenascin family member tenascin-N and conditional gene targeting of tenascin-R
Elimination of the four extracellular matrix molecules tenascin-C, tenascin-R, brevican and neurocan alters the ratio of excitatory and inhibitory synapses
The extracellular matrix proteins tenascin-C and tenascin-R retard oligodendrocyte precursor maturation and myelin regeneration in a cuprizone-induced long-term demyelination animal model
Brevican, Neurocan, Tenascin-C, and Tenascin-R act as important regulators of the interplay between perineuronal nets, synaptic integrity, inhibitory interneurons, and Otx2
Substrate-bound and soluble domains of tenascin-C regulate differentiation, proliferation and migration of neural stem and progenitor cells
Cell tracking in vitro reveals that the extracellular matrix glycoprotein Tenascin-C
Role of the extracellular matrix glycoprotein Tenascin-c in brain ischemia and ist regulation by Erythropoietin
Modulation of GluN3B-containing excitatory glycine receptors by zinc
Low-density lipoprotein receptor-related protein-1 (LRP1) in the glial lineage modulates neuronal excitability
The guanine nucleotide exchange factor Vav3 intervenes in the migration pathway of oligodendrocyte precursor cells on tenascin-C
Cell tracking in vitro reveals that the extracellular matrix glycoprotein Tenascin-C modulates cell cycle length and differentiation in neural stem/progenitor cells of the developing mouse spinal cord
Loss of the extracellular matrix molecule tenascin-C leads to absence of reactive gliosis and promotes anti-inflammatory cytokine expression in an autoimmune glaucoma mouse model