Prolactin-induced mouse mammary carcinomas model estrogen resistant luminal breast cancer
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1465-542X
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
-
Prolactin-induced mouse mammary carcinomas model estrogen resistant luminal breast cancer ; volume:13 ; number:1 ; day:28 ; month:1 ; year:2011 ; pages:1-15 ; date:2.2011
Breast cancer research ; 13, Heft 1 (28.1.2011), 1-15, 2.2011
- Creator
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Arendt, Lisa M.
- Contributor
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Rugowski, Debra E.
Grafwallner-Huseth, Tara A.
Garcia-Barchino, Maria Jose
Rui, Hallgeir
Schuler, Linda A.
SpringerLink (Online service)
- DOI
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10.1186/bcr2819
- URN
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urn:nbn:de:1111-2016032411416
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:50 AM CEST
Data provider
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Associated
- Arendt, Lisa M.
- Rugowski, Debra E.
- Grafwallner-Huseth, Tara A.
- Garcia-Barchino, Maria Jose
- Rui, Hallgeir
- Schuler, Linda A.
- SpringerLink (Online service)
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STAT1-deficient mice spontaneously develop estrogen receptor α-positive luminal mammary carcinomas
Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas
Expression of prolactin receptors in normal canine mammary tissue, canine mammary adenomas and mammary adenocarcinomas
MRI features of breast cancer immunophenotypes with a focus on luminal estrogen receptor low positive invasive carcinomas
Androgen receptor and FOXA1 coexpression define a “luminal-AR” subtype of feline mammary carcinomas, spontaneous models of breast cancer
Elevated collagen-I augments tumor progressive signals, intravasation and metastasis of prolactin-induced estrogen receptor alpha positive mammary tumor cells
Multipotent luminal mammary cancer stem cells model tumor heterogeneity
Estrogen receptors genotypes and canine mammary neoplasia
Cancer-associated fibroblasts rewire the estrogen receptor response in luminal breast cancer, enabling estrogen independence
Estrogen-induced immune changes within the normal mammary gland
VANGL2 regulates luminal epithelial organization and cell turnover in the mammary gland
The role of Sox9 in mouse mammary gland development and maintenance of mammary stem and luminal progenitor cells
STAT1-deficient mice spontaneously develop estrogen receptor α-positive luminal mammary carcinomas
Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas
Expression of prolactin receptors in normal canine mammary tissue, canine mammary adenomas and mammary adenocarcinomas
MRI features of breast cancer immunophenotypes with a focus on luminal estrogen receptor low positive invasive carcinomas
Androgen receptor and FOXA1 coexpression define a “luminal-AR” subtype of feline mammary carcinomas, spontaneous models of breast cancer
Elevated collagen-I augments tumor progressive signals, intravasation and metastasis of prolactin-induced estrogen receptor alpha positive mammary tumor cells
Multipotent luminal mammary cancer stem cells model tumor heterogeneity
Estrogen receptors genotypes and canine mammary neoplasia
Cancer-associated fibroblasts rewire the estrogen receptor response in luminal breast cancer, enabling estrogen independence
Estrogen-induced immune changes within the normal mammary gland
VANGL2 regulates luminal epithelial organization and cell turnover in the mammary gland