Mesenchymal stem cell repression of Th17 cells is triggered by mitochondrial transfer
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1757-6512
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Mesenchymal stem cell repression of Th17 cells is triggered by mitochondrial transfer ; volume:10 ; number:1 ; day:1 ; month:8 ; year:2019 ; pages:1-13 ; date:12.2019
Stem cell research & therapy ; 10, Heft 1 (1.8.2019), 1-13, 12.2019
- Creator
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Luz-Crawford, Patricia
Hernandez, Javier
Djouad, Farida
Luque-Campos, Noymar
Caicedo, Andres
Carrère-Kremer, Séverine
Brondello, Jean-Marc
Vignais, Marie-Luce
Pène, Jérôme
Jorgensen, Christian
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s13287-019-1307-9
- URN
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urn:nbn:de:101:1-2019122603153410545001
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:57 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Luz-Crawford, Patricia
- Hernandez, Javier
- Djouad, Farida
- Luque-Campos, Noymar
- Caicedo, Andres
- Carrère-Kremer, Séverine
- Brondello, Jean-Marc
- Vignais, Marie-Luce
- Pène, Jérôme
- Jorgensen, Christian
- SpringerLink (Online service)
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Mesenchymal stem cells regulate the Th17/Treg cell balance partly through hepatocyte growth factor in vitro
Mesenchymal stromal cells restrain the Th17 cell response via L-amino-acid oxidase within lymph nodes
TGF-β1-overexpressing mesenchymal stem cells reciprocally regulate Th17/Treg cells by regulating the expression of IFN-γ
Mesenchymal stem cells generate a CD4+CD25+Foxp3+ regulatory T cell population during the differentiation process of Th1 and Th17 cells
Impaired autophagy triggered by HDAC9 in mesenchymal stem cells accelerates bone mass loss
Altered T cell plasticity favors Th17 cells in rheumatoid arthritis
Spliceosomal repression: unleashing human cell totipotency
CAR modified T cells resist TGF-b mediated repression through engineered IL-7 triggered IL-2 signaling
Th17 cells
SOST Gene Inhibits Osteogenesis from Adipose-Derived Mesenchymal Stem Cells by Inducing Th17 Cell Differentiation
Reduced graphene oxide triggered epithelial-mesenchymal transition in A549 cells
Mesenchymal stem cells repress Th17 molecular program through the PD-1 pathway
Mesenchymal stem cells regulate the Th17/Treg cell balance partly through hepatocyte growth factor in vitro
Mesenchymal stromal cells restrain the Th17 cell response via L-amino-acid oxidase within lymph nodes
TGF-β1-overexpressing mesenchymal stem cells reciprocally regulate Th17/Treg cells by regulating the expression of IFN-γ
Mesenchymal stem cells generate a CD4+CD25+Foxp3+ regulatory T cell population during the differentiation process of Th1 and Th17 cells
Impaired autophagy triggered by HDAC9 in mesenchymal stem cells accelerates bone mass loss
Altered T cell plasticity favors Th17 cells in rheumatoid arthritis
Spliceosomal repression: unleashing human cell totipotency
CAR modified T cells resist TGF-b mediated repression through engineered IL-7 triggered IL-2 signaling
Th17 cells
SOST Gene Inhibits Osteogenesis from Adipose-Derived Mesenchymal Stem Cells by Inducing Th17 Cell Differentiation
Reduced graphene oxide triggered epithelial-mesenchymal transition in A549 cells
Mesenchymal stem cells repress Th17 molecular program through the PD-1 pathway
Mesenchymal stem cells regulate the Th17/Treg cell balance partly through hepatocyte growth factor in vitro
Mesenchymal stromal cells restrain the Th17 cell response via L-amino-acid oxidase within lymph nodes
TGF-β1-overexpressing mesenchymal stem cells reciprocally regulate Th17/Treg cells by regulating the expression of IFN-γ
Mesenchymal stem cells generate a CD4+CD25+Foxp3+ regulatory T cell population during the differentiation process of Th1 and Th17 cells
Impaired autophagy triggered by HDAC9 in mesenchymal stem cells accelerates bone mass loss
Altered T cell plasticity favors Th17 cells in rheumatoid arthritis
Spliceosomal repression: unleashing human cell totipotency