Smac/DIABLO enhances the therapeutic potential of chemotherapeutic drugs and irradiation, and sensitizes TRAIL-resistant breast cancer cells
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1476-4598
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Smac/DIABLO enhances the therapeutic potential of chemotherapeutic drugs and irradiation, and sensitizes TRAIL-resistant breast cancer cells ; volume:7 ; number:1 ; day:30 ; month:6 ; year:2008 ; pages:1-10 ; date:12.2008
Molecular cancer ; 7, Heft 1 (30.6.2008), 1-10, 12.2008
- Creator
-
Fandy, Tamer E.
Shankar, Sharmila
Srivastava, Rakesh K.
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1186/1476-4598-7-60
- URN
-
urn:nbn:de:101:1-2021080822305447807047
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:58 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Fandy, Tamer E.
- Shankar, Sharmila
- Srivastava, Rakesh K.
- SpringerLink (Online service)
Other Objects (12)
Role of Smac/DIABLO in cancer progression
Targeting bone morphogenetic protein receptor 2 sensitizes lung cancer cells to TRAIL by increasing cytosolic Smac/DIABLO and the downregulation of X-linked inhibitor of apoptosis protein
Mechanismen der Apoptoseinduktion durch das proapoptotische Protein Smac/DIABLO
Mechanismen der Apoptoseinduktion durch das proapoptotische Protein Smac/DIABLO
A Smac-mimetic sensitizes prostate cancer cells to TRAIL-induced apoptosis via modulating both IAPs and NF-kappaB
Smac mimetic-derived augmentation of chemotherapeutic response in experimental pancreatic cancer
Bortezomib sensitizes neuroblastoma for TRAIL-induced apoptosis
High Smac/DIABLO expression is associated with early local recurrence of cervical cancer
UHRF1 depletion sensitizes retinoblastoma cells to chemotherapeutic drugs via downregulation of XRCC4
Smac/DIABLO protein acts as an independent prognostic factor in oral squamous cell carcinoma
Curcumin sensitizes TRAIL-resistant xenografts: molecular mechanisms of apoptosis, metastasis and angiogenesis
Prognostic significance of pretreatment VEGF, survivin, and Smac/DIABLO serum levels in patients with serous ovarian carcinoma
Role of Smac/DIABLO in cancer progression
Targeting bone morphogenetic protein receptor 2 sensitizes lung cancer cells to TRAIL by increasing cytosolic Smac/DIABLO and the downregulation of X-linked inhibitor of apoptosis protein
Mechanismen der Apoptoseinduktion durch das proapoptotische Protein Smac/DIABLO
Mechanismen der Apoptoseinduktion durch das proapoptotische Protein Smac/DIABLO
A Smac-mimetic sensitizes prostate cancer cells to TRAIL-induced apoptosis via modulating both IAPs and NF-kappaB
Smac mimetic-derived augmentation of chemotherapeutic response in experimental pancreatic cancer
Bortezomib sensitizes neuroblastoma for TRAIL-induced apoptosis
High Smac/DIABLO expression is associated with early local recurrence of cervical cancer
UHRF1 depletion sensitizes retinoblastoma cells to chemotherapeutic drugs via downregulation of XRCC4
Smac/DIABLO protein acts as an independent prognostic factor in oral squamous cell carcinoma
Curcumin sensitizes TRAIL-resistant xenografts: molecular mechanisms of apoptosis, metastasis and angiogenesis
Prognostic significance of pretreatment VEGF, survivin, and Smac/DIABLO serum levels in patients with serous ovarian carcinoma
Role of Smac/DIABLO in cancer progression
Targeting bone morphogenetic protein receptor 2 sensitizes lung cancer cells to TRAIL by increasing cytosolic Smac/DIABLO and the downregulation of X-linked inhibitor of apoptosis protein
Mechanismen der Apoptoseinduktion durch das proapoptotische Protein Smac/DIABLO
Mechanismen der Apoptoseinduktion durch das proapoptotische Protein Smac/DIABLO
A Smac-mimetic sensitizes prostate cancer cells to TRAIL-induced apoptosis via modulating both IAPs and NF-kappaB
Smac mimetic-derived augmentation of chemotherapeutic response in experimental pancreatic cancer
Bortezomib sensitizes neuroblastoma for TRAIL-induced apoptosis
High Smac/DIABLO expression is associated with early local recurrence of cervical cancer
UHRF1 depletion sensitizes retinoblastoma cells to chemotherapeutic drugs via downregulation of XRCC4
Smac/DIABLO protein acts as an independent prognostic factor in oral squamous cell carcinoma
Curcumin sensitizes TRAIL-resistant xenografts: molecular mechanisms of apoptosis, metastasis and angiogenesis