Sociability deficits after prenatal exposure to valproic acid are rescued by early social enrichment
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2040-2392
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
-
Sociability deficits after prenatal exposure to valproic acid are rescued by early social enrichment ; volume:9 ; number:1 ; day:14 ; month:6 ; year:2018 ; pages:1-17 ; date:12.2018
Molecular autism ; 9, Heft 1 (14.6.2018), 1-17, 12.2018
- Creator
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Campolongo, Marcos
- Contributor
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Kazlauskas, Nadia
Falasco, German
Urrutia, Leandro
Salgueiro, Natalí
Höcht, Christian
Depino, Amaicha Mara
SpringerLink (Online service)
- DOI
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10.1186/s13229-018-0221-9
- URN
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urn:nbn:de:101:1-2018082219041534876423
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:33 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Campolongo, Marcos
- Kazlauskas, Nadia
- Falasco, German
- Urrutia, Leandro
- Salgueiro, Natalí
- Höcht, Christian
- Depino, Amaicha Mara
- SpringerLink (Online service)
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Gastrointestinal Tract Abnormalities Induced by Prenatal Valproic Acid Exposure in Rat Offspring
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Deficits in integrative NMDA receptors caused by Grin1 disruption can be rescued in adulthood
Prenatal Stress Induces Translational Disruption Associated with Myelination Deficits
Prenatal exposure to valproic acid increases miR-132 levels in the mouse embryonic brain
Huntington disease oligodendrocyte maturation deficits revealed by single-nucleus RNAseq are rescued by thiamine-biotin supplementation
Transient CSF1R inhibition ameliorates behavioral deficits in Cntnap2 knockout and valproic acid-exposed mouse models of autism
Prenatal chlorpyrifos leads to autism-like deficits in C57Bl6/J mice
Cariprazine alleviates core behavioral deficits in the prenatal valproic acid exposure model of autism spectrum disorder
Rescued
Rescued!
Rescued!
Gastrointestinal Tract Abnormalities Induced by Prenatal Valproic Acid Exposure in Rat Offspring
Consequences of prenatal exposure to valproic acid in the socially monogamous prairie voles
Deficits in integrative NMDA receptors caused by Grin1 disruption can be rescued in adulthood
Prenatal Stress Induces Translational Disruption Associated with Myelination Deficits
Prenatal exposure to valproic acid increases miR-132 levels in the mouse embryonic brain
Huntington disease oligodendrocyte maturation deficits revealed by single-nucleus RNAseq are rescued by thiamine-biotin supplementation
Transient CSF1R inhibition ameliorates behavioral deficits in Cntnap2 knockout and valproic acid-exposed mouse models of autism