PI3Kδ activity controls plasticity and discriminates between EMT and stemness based on distinct TGFβ signaling
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2399-3642
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
PI3Kδ activity controls plasticity and discriminates between EMT and stemness based on distinct TGFβ signaling ; volume:5 ; number:1 ; day:25 ; month:7 ; year:2022 ; pages:1-15 ; date:12.2022
Communications biology ; 5, Heft 1 (25.7.2022), 1-15, 12.2022
- Creator
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Agnetti, Jean
Bou Malham, Vanessa
Desterke, Christophe
Benzoubir, Nassima
Peng, Juan
Jacques, Sophie
Rahmouni, Souad
Valentin, Emanuel Di
Tan, Tuan Zea
Samuel, Didier
Thiery, Jean Paul
Gassama-Diagne, Ama
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s42003-022-03637-w
- URN
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urn:nbn:de:101:1-2022100619412522886580
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:28 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Agnetti, Jean
- Bou Malham, Vanessa
- Desterke, Christophe
- Benzoubir, Nassima
- Peng, Juan
- Jacques, Sophie
- Rahmouni, Souad
- Valentin, Emanuel Di
- Tan, Tuan Zea
- Samuel, Didier
- Thiery, Jean Paul
- Gassama-Diagne, Ama
- SpringerLink (Online service)
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The dynamic interplay of ZEB1, CD44 and HAS2 promotes EMT and stemness in cancer
The ZEB1-miR-200 feedback loop controls the Notch signaling pathway and alternative splicing to regulate EMT and stemness
KMT9 controls stemness and growth of colorectal cancer
TGF-β isoforms induce EMT independent migration of ovarian cancer cells
Discovery of a natural small-molecule compound that suppresses tumor EMT, stemness and metastasis by inhibiting TGFβ/BMP signaling in triple-negative breast cancer
Chemotherapeutic stress influences EMT and stemness in cancer stem cells of TNBC
How BMP-2 induces EMT and breast cancer stemness through Rb and CD44?
Loss of Stemness, EMT, and Supernumerary Tooth Formation in Cebpb −/− Runx2 +/− Murine Incisors
TGFβ signaling networks in ovarian cancer progression and plasticity
DNA methylation controls stemness of astrocytes in health and ischaemia
TGFβ-induzierte [TGF-beta-induzierte] epithelial-mesenchymale Transformation (EMT) und Zunahme der Invasivität in TGFβ-responsiven [TGF-beta-responsiven] Pankreaskarzinomzellinien
EMT and stemness: flexible processes tuned by alternative splicing in development and cancer progression
The dynamic interplay of ZEB1, CD44 and HAS2 promotes EMT and stemness in cancer
The ZEB1-miR-200 feedback loop controls the Notch signaling pathway and alternative splicing to regulate EMT and stemness
KMT9 controls stemness and growth of colorectal cancer
TGF-β isoforms induce EMT independent migration of ovarian cancer cells
Discovery of a natural small-molecule compound that suppresses tumor EMT, stemness and metastasis by inhibiting TGFβ/BMP signaling in triple-negative breast cancer
Chemotherapeutic stress influences EMT and stemness in cancer stem cells of TNBC
How BMP-2 induces EMT and breast cancer stemness through Rb and CD44?
Loss of Stemness, EMT, and Supernumerary Tooth Formation in Cebpb −/− Runx2 +/− Murine Incisors
TGFβ signaling networks in ovarian cancer progression and plasticity
DNA methylation controls stemness of astrocytes in health and ischaemia
TGFβ-induzierte [TGF-beta-induzierte] epithelial-mesenchymale Transformation (EMT) und Zunahme der Invasivität in TGFβ-responsiven [TGF-beta-responsiven] Pankreaskarzinomzellinien
EMT and stemness: flexible processes tuned by alternative splicing in development and cancer progression