ACE2 deficiency exacerbates obesity-related glomerulopathy through its role in regulating lipid metabolism

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2058-7716

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: ACE2 deficiency exacerbates obesity-related glomerulopathy through its role in regulating lipid metabolism ; volume:8 ; number:1 ; day:30 ; month:9 ; year:2022 ; pages:1-9 ; date:12.2022
Cell death discovery ; 8, Heft 1 (30.9.2022), 1-9, 12.2022

Creator: Che, Yin-Yin
Hong, Han
Lei, Yu-Ting
Zou, Jia
Yang, Yi-Ya
He, Li-Yu

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41420-022-01191-2

URN: urn:nbn:de:101:1-2022121809035141281762

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:36 AM CEST

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Che, Yin-Yin
Hong, Han
Lei, Yu-Ting
Zou, Jia
Yang, Yi-Ya
He, Li-Yu
SpringerLink (Online service)

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Correction: ACE2 deficiency exacerbates obesity-related glomerulopathy through its role in regulating lipid metabolism

MATE1 Deficiency Exacerbates Dofetilide-Induced Proarrhythmia

Leptin/obR signaling exacerbates obesity-related neutrophilic airway inflammation through inflammatory M1 macrophages

Kallistatin deficiency exacerbates neuronal damage after cardiac arrest

Interleukin-10 deficiency exacerbates inflammation-induced tau pathology

Ageing, ACE2 deficiency and bad outcome in COVID-19

Complement C6 deficiency exacerbates pathophysiology after spinal cord injury

SARS-CoV-2 Spike Protein Exacerbates Thromboembolic Cerebrovascular Complications in Humanized ACE2 Mouse Model

Interleukin-6 deficiency exacerbates Huntington’s disease model phenotypes

Dystrophin deficiency exacerbates skeletal muscle pathology in dysferlin-null mice

Parkin deficiency exacerbates fasting-induced skeletal muscle wasting in mice

TAZ deficiency exacerbates psoriatic pathogenesis by increasing the histamine-releasing factor

Correction: ACE2 deficiency exacerbates obesity-related glomerulopathy through its role in regulating lipid metabolism

MATE1 Deficiency Exacerbates Dofetilide-Induced Proarrhythmia

Leptin/obR signaling exacerbates obesity-related neutrophilic airway inflammation through inflammatory M1 macrophages

Kallistatin deficiency exacerbates neuronal damage after cardiac arrest

Interleukin-10 deficiency exacerbates inflammation-induced tau pathology

Ageing, ACE2 deficiency and bad outcome in COVID-19

Complement C6 deficiency exacerbates pathophysiology after spinal cord injury

SARS-CoV-2 Spike Protein Exacerbates Thromboembolic Cerebrovascular Complications in Humanized ACE2 Mouse Model

Interleukin-6 deficiency exacerbates Huntington’s disease model phenotypes

Dystrophin deficiency exacerbates skeletal muscle pathology in dysferlin-null mice

Parkin deficiency exacerbates fasting-induced skeletal muscle wasting in mice

TAZ deficiency exacerbates psoriatic pathogenesis by increasing the histamine-releasing factor

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ACE2 deficiency exacerbates obesity-related glomerulopathy through its role in regulating lipid metabolism

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Other Objects (12)

Correction: ACE2 deficiency exacerbates obesity-related glomerulopathy through its role in regulating lipid metabolism

MATE1 Deficiency Exacerbates Dofetilide-Induced Proarrhythmia

Leptin/obR signaling exacerbates obesity-related neutrophilic airway inflammation through inflammatory M1 macrophages

Kallistatin deficiency exacerbates neuronal damage after cardiac arrest

Interleukin-10 deficiency exacerbates inflammation-induced tau pathology

Ageing, ACE2 deficiency and bad outcome in COVID-19

Complement C6 deficiency exacerbates pathophysiology after spinal cord injury

SARS-CoV-2 Spike Protein Exacerbates Thromboembolic Cerebrovascular Complications in Humanized ACE2 Mouse Model

Interleukin-6 deficiency exacerbates Huntington’s disease model phenotypes

Dystrophin deficiency exacerbates skeletal muscle pathology in dysferlin-null mice

Parkin deficiency exacerbates fasting-induced skeletal muscle wasting in mice

TAZ deficiency exacerbates psoriatic pathogenesis by increasing the histamine-releasing factor

Correction: ACE2 deficiency exacerbates obesity-related glomerulopathy through its role in regulating lipid metabolism

MATE1 Deficiency Exacerbates Dofetilide-Induced Proarrhythmia

Leptin/obR signaling exacerbates obesity-related neutrophilic airway inflammation through inflammatory M1 macrophages

Kallistatin deficiency exacerbates neuronal damage after cardiac arrest

Interleukin-10 deficiency exacerbates inflammation-induced tau pathology

Ageing, ACE2 deficiency and bad outcome in COVID-19

Complement C6 deficiency exacerbates pathophysiology after spinal cord injury

SARS-CoV-2 Spike Protein Exacerbates Thromboembolic Cerebrovascular Complications in Humanized ACE2 Mouse Model

Interleukin-6 deficiency exacerbates Huntington’s disease model phenotypes

Dystrophin deficiency exacerbates skeletal muscle pathology in dysferlin-null mice

Parkin deficiency exacerbates fasting-induced skeletal muscle wasting in mice

TAZ deficiency exacerbates psoriatic pathogenesis by increasing the histamine-releasing factor

Correction: ACE2 deficiency exacerbates obesity-related glomerulopathy through its role in regulating lipid metabolism

MATE1 Deficiency Exacerbates Dofetilide-Induced Proarrhythmia

Leptin/obR signaling exacerbates obesity-related neutrophilic airway inflammation through inflammatory M1 macrophages

Kallistatin deficiency exacerbates neuronal damage after cardiac arrest

Interleukin-10 deficiency exacerbates inflammation-induced tau pathology

Ageing, ACE2 deficiency and bad outcome in COVID-19

Complement C6 deficiency exacerbates pathophysiology after spinal cord injury

SARS-CoV-2 Spike Protein Exacerbates Thromboembolic Cerebrovascular Complications in Humanized ACE2 Mouse Model

Interleukin-6 deficiency exacerbates Huntington’s disease model phenotypes

Dystrophin deficiency exacerbates skeletal muscle pathology in dysferlin-null mice

Parkin deficiency exacerbates fasting-induced skeletal muscle wasting in mice

TAZ deficiency exacerbates psoriatic pathogenesis by increasing the histamine-releasing factor

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