ROS-independent ER stress-mediated NRF2 activation promotes warburg effect to maintain stemness-associated properties of cancer-initiating cells
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2041-4889
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
-
ROS-independent ER stress-mediated NRF2 activation promotes warburg effect to maintain stemness-associated properties of cancer-initiating cells ; volume:9 ; number:2 ; day:7 ; month:2 ; year:2018 ; pages:1-14 ; date:2.2018
Cell death & disease ; 9, Heft 2 (7.2.2018), 1-14, 2.2018
- Creator
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Chang, Ching-Wen
- Contributor
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Chen, Yu-Syuan
Tsay, Yeou-Guang
Han, Chia-Li
Chen, Yu-Ju
Yang, Cheng-Chieh
Hung, Kai-Feng
Lin, Chao-Hsiung
Huang, Tsung-Yen
Kao, Shou-Yen
Lee, Te-Chang
Lo, Jeng-Fan
SpringerLink (Online service)
- DOI
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10.1038/s41419-017-0250-x
- URN
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urn:nbn:de:101:1-2018090801532286047740
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:21 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Chang, Ching-Wen
- Chen, Yu-Syuan
- Tsay, Yeou-Guang
- Han, Chia-Li
- Chen, Yu-Ju
- Yang, Cheng-Chieh
- Hung, Kai-Feng
- Lin, Chao-Hsiung
- Huang, Tsung-Yen
- Kao, Shou-Yen
- Lee, Te-Chang
- Lo, Jeng-Fan
- SpringerLink (Online service)
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PIWIL2/PDK1 Axis Promotes the Progression of Cervical Epithelial Lesions via Metabolic Reprogramming to Maintain Tumor‐Initiating Cell Stemness
Oocytes maintain ROS-free mitochondrial metabolism by suppressing complex I
Nrf2 is required to maintain the self-renewal of glioma stem cells
R-spodin2 enhances canonical Wnt signaling to maintain the stemness of glioblastoma cells
Correction to: Nrf2 is required to maintain the self-renewal of glioma stem cells
Enhancer remodeling promotes tumor-initiating activity in NRF2-activated non-small cell lung cancers
Mouse embryonic palatal mesenchymal cells maintain stemness through the PTEN-Akt-mTOR autophagic pathway
NLK functions to maintain proliferation and stemness of NSCLC and is a target of metformin
Exogenous H2S initiating Nrf2/GPx4/GSH pathway through promoting Syvn1-Keap1 interaction in diabetic hearts
Erratum to: NLK functions to maintain proliferation and stemness of NSCLC and is a target of metformin
The role of the SWI/SNF chromatin remodeling complex in maintaining the stemness of glioma initiating cells
KAT6A regulates stemness of aging bone marrow-derived mesenchymal stem cells through Nrf2/ARE signaling pathway
PIWIL2/PDK1 Axis Promotes the Progression of Cervical Epithelial Lesions via Metabolic Reprogramming to Maintain Tumor‐Initiating Cell Stemness
Oocytes maintain ROS-free mitochondrial metabolism by suppressing complex I
Nrf2 is required to maintain the self-renewal of glioma stem cells
R-spodin2 enhances canonical Wnt signaling to maintain the stemness of glioblastoma cells
Correction to: Nrf2 is required to maintain the self-renewal of glioma stem cells
Enhancer remodeling promotes tumor-initiating activity in NRF2-activated non-small cell lung cancers
Mouse embryonic palatal mesenchymal cells maintain stemness through the PTEN-Akt-mTOR autophagic pathway
NLK functions to maintain proliferation and stemness of NSCLC and is a target of metformin
Exogenous H2S initiating Nrf2/GPx4/GSH pathway through promoting Syvn1-Keap1 interaction in diabetic hearts
Erratum to: NLK functions to maintain proliferation and stemness of NSCLC and is a target of metformin
The role of the SWI/SNF chromatin remodeling complex in maintaining the stemness of glioma initiating cells