Neurotoxic Microglial Activation via IFNγ‐Induced Nrf2 Reduction Exacerbating Alzheimer's Disease (Adv. Sci. 20/2024)
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Bibliographic citation
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Neurotoxic Microglial Activation via IFNγ‐Induced Nrf2 Reduction Exacerbating Alzheimer's Disease (Adv. Sci. 20/2024) ; volume:11 ; number:20 ; year:2024 ; extent:1
Advanced science ; 11, Heft 20 (2024) (gesamt 1)
- Creator
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Kang, You Jung
Hyeon, Seung Jae
McQuade, Amanda
Lim, Jiwoon
Baek, Seung Hyun
Diep, Yen N.
Do, Khanh V.
Jeon, Yeji
Jo, Dong‐Gyu
Lee, C. Justin
Blurton‐Jones, Mathew
Ryu, Hoon
Cho, Hansang
- DOI
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10.1002/advs.202470114
- URN
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urn:nbn:de:101:1-2405291419040.140441223348
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:47 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Kang, You Jung
- Hyeon, Seung Jae
- McQuade, Amanda
- Lim, Jiwoon
- Baek, Seung Hyun
- Diep, Yen N.
- Do, Khanh V.
- Jeon, Yeji
- Jo, Dong‐Gyu
- Lee, C. Justin
- Blurton‐Jones, Mathew
- Ryu, Hoon
- Cho, Hansang
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Hippocampal microglial activation triggers a neurotoxic-specific astrocyte response and mediates etomidate-induced long-term synaptic inhibition
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Parkinson’s disease-derived α-synuclein assemblies combined with chronic-type inflammatory cues promote a neurotoxic microglial phenotype
Plasma microglial-derived extracellular vesicles are increased in frail patients with Mild Cognitive Impairment and exert a neurotoxic effect
Apomorphine Reduces A53T α-Synuclein-Induced Microglial Reactivity Through Activation of NRF2 Signalling Pathway
EGF/STAT1-maintained ECM1 expression in hepatic homeostasis is disrupted by IFNγ/NRF2 in chronic liver diseases
Prevention of neurotoxic illness in working populations
Neurotoxic effect of antiretroviral agents on CNS
Neurotoxic Microglial Activation via IFNγ‐Induced Nrf2 Reduction Exacerbating Alzheimer's Disease
The NALP3 inflammasome is involved in neurotoxic prion peptide-induced microglial activation
Selective GSK3β Inhibition Mediates an Nrf2-Independent Anti-inflammatory Microglial Response
Salsolinol—neurotoxic or Neuroprotective?
Hippocampal microglial activation triggers a neurotoxic-specific astrocyte response and mediates etomidate-induced long-term synaptic inhibition
Gastrointestinal Incomplete Degradation Exacerbates Neurotoxic Effects of PLA Microplastics via Oligomer Nanoplastics Formation (Adv. Sci. 28/2024)
Parkinson’s disease-derived α-synuclein assemblies combined with chronic-type inflammatory cues promote a neurotoxic microglial phenotype
Plasma microglial-derived extracellular vesicles are increased in frail patients with Mild Cognitive Impairment and exert a neurotoxic effect
Apomorphine Reduces A53T α-Synuclein-Induced Microglial Reactivity Through Activation of NRF2 Signalling Pathway
EGF/STAT1-maintained ECM1 expression in hepatic homeostasis is disrupted by IFNγ/NRF2 in chronic liver diseases
Prevention of neurotoxic illness in working populations
Neurotoxic effect of antiretroviral agents on CNS
Neurotoxic Microglial Activation via IFNγ‐Induced Nrf2 Reduction Exacerbating Alzheimer's Disease
The NALP3 inflammasome is involved in neurotoxic prion peptide-induced microglial activation
Selective GSK3β Inhibition Mediates an Nrf2-Independent Anti-inflammatory Microglial Response
Salsolinol—neurotoxic or Neuroprotective?
Hippocampal microglial activation triggers a neurotoxic-specific astrocyte response and mediates etomidate-induced long-term synaptic inhibition
Gastrointestinal Incomplete Degradation Exacerbates Neurotoxic Effects of PLA Microplastics via Oligomer Nanoplastics Formation (Adv. Sci. 28/2024)
Parkinson’s disease-derived α-synuclein assemblies combined with chronic-type inflammatory cues promote a neurotoxic microglial phenotype
Plasma microglial-derived extracellular vesicles are increased in frail patients with Mild Cognitive Impairment and exert a neurotoxic effect
Apomorphine Reduces A53T α-Synuclein-Induced Microglial Reactivity Through Activation of NRF2 Signalling Pathway
EGF/STAT1-maintained ECM1 expression in hepatic homeostasis is disrupted by IFNγ/NRF2 in chronic liver diseases
Prevention of neurotoxic illness in working populations