- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
-
Englisch
- Bibliographic citation
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Candida albicans Virulence Traits in Commensalism and Disease ; volume:11 ; number:4 ; day:1 ; month:10 ; year:2024 ; pages:231-240 ; date:12.2024
Current clinical microbiology reports ; 11, Heft 4 (1.10.2024), 231-240, 12.2024
- Creator
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Fróis-Martins, Ricardo
Lagler, Julia
LeibundGut-Landmann, Salomé
- Contributor
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SpringerLink (Online service)
- DOI
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10.1007/s40588-024-00235-8
- URN
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urn:nbn:de:101:1-2501242118585.118596131635
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:37 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Fróis-Martins, Ricardo
- Lagler, Julia
- LeibundGut-Landmann, Salomé
- SpringerLink (Online service)
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Candida albicans virulence and drug-resistance requires the O-acyltransferase Gup1p
Virulence and in vitro antifungal susceptibility of Candida albicans and Candida catenulata from laying hens
Nutrient acquisition and metabolic adaptation in the context of Candida albicans virulence
Stoffwechselstudien an Candida albicans
Virulence-Related Genes Expression in Planktonic Mixed Cultures of Candida a lbicans and Non-Albicans Candida Species
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Mannich base limits Candida albicans virulence by inactivating Ras-cAMP-PKA pathway
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Control of nitrogen regulated virulence traits of the human fungal pathogen Candida albicans : = Steuerung von stickstoffregulierten Virulenzeigenschaften des human-pathogenen Pilzes Candida albicans
Regulation of virulence-associated traits of the human fungal pathogen Candida albicans by nitrogen availability
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Virulence-Related Genes Expression in Planktonic Mixed Cultures of Candida a lbicans and Non-Albicans Candida Species
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Mannich base limits Candida albicans virulence by inactivating Ras-cAMP-PKA pathway
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