Glycine-alanine dipeptide repeat protein contributes to toxicity in a zebrafish model of C9orf72 associated neurodegeneration

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1750-1326

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Glycine-alanine dipeptide repeat protein contributes to toxicity in a zebrafish model of C9orf72 associated neurodegeneration ; volume:12 ; number:1 ; day:14 ; month:1 ; year:2017 ; pages:1-11 ; date:12.2017
Molecular neurodegeneration ; 12, Heft 1 (14.1.2017), 1-11, 12.2017

Classification: Biowissenschaften, Biologie

Creator: Ohki, Yu

Contributor: Wenninger-Weinzierl, Andrea
Hruscha, Alexander
Asakawa, Kazuhide
Kawakami, Koichi
Haass, Christian
Edbauer, Dieter
Schmid, Bettina
SpringerLink (Online service)

DOI: 10.1186/s13024-016-0146-8

URN: urn:nbn:de:1111-201702123952

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:50 AM CEST

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Associated

Ohki, Yu
Wenninger-Weinzierl, Andrea
Hruscha, Alexander
Asakawa, Kazuhide
Kawakami, Koichi
Haass, Christian
Edbauer, Dieter
Schmid, Bettina
SpringerLink (Online service)

Other Objects (12)

Repeat length of C9orf72-associated glycine–alanine polypeptides affects their toxicity

Poly-GR dipeptide repeat polymers correlate with neurodegeneration and Clinicopathological subtypes in C9ORF72-related brain disease

Investigating the spread and toxicity of glycine-alanine dipeptides in C9orf72 ALS/FTD using Drosophila melanogaster

Synaptic dysfunction induced by glycine‐alanine dipeptides in C9orf72‐ ALS/FTD is rescued by SV 2 replenishment

Glycine-alanine dipeptide repeats spread rapidly in a repeat length- and age-dependent manner in the fly brain

CRISPR deletion of the C9ORF72 promoter in ALS/FTD patient motor neurons abolishes production of dipeptide repeat proteins and rescues neurodegeneration

Antibodies inhibit transmission and aggregation of C9orf72 poly‐ GA dipeptide repeat proteins

Poly-glycine–alanine exacerbates C9orf72 repeat expansion-mediated DNA damage via sequestration of phosphorylated ATM and loss of nuclear hnRNPA3

Hochschulschrift

Characterization of the C9orf72 dipeptide repeat proteins in frontotemporal dementia and amyotrophic lateral sclerosis

Dipeptide repeat proteins activate a heat shock response found in C9ORF72-ALS/FTLD patients

Dipeptide repeat (DPR) pathology in the skeletal muscle of ALS patients with C9ORF72 repeat expansion

CUG initiation and frameshifting enable production of dipeptide repeat proteins from ALS/FTD C9ORF72 transcripts

Repeat length of C9orf72-associated glycine–alanine polypeptides affects their toxicity

Poly-GR dipeptide repeat polymers correlate with neurodegeneration and Clinicopathological subtypes in C9ORF72-related brain disease

Investigating the spread and toxicity of glycine-alanine dipeptides in C9orf72 ALS/FTD using Drosophila melanogaster

Synaptic dysfunction induced by glycine‐alanine dipeptides in C9orf72‐ ALS/FTD is rescued by SV 2 replenishment

Glycine-alanine dipeptide repeats spread rapidly in a repeat length- and age-dependent manner in the fly brain

CRISPR deletion of the C9ORF72 promoter in ALS/FTD patient motor neurons abolishes production of dipeptide repeat proteins and rescues neurodegeneration

Antibodies inhibit transmission and aggregation of C9orf72 poly‐ GA dipeptide repeat proteins

Poly-glycine–alanine exacerbates C9orf72 repeat expansion-mediated DNA damage via sequestration of phosphorylated ATM and loss of nuclear hnRNPA3

Hochschulschrift

Characterization of the C9orf72 dipeptide repeat proteins in frontotemporal dementia and amyotrophic lateral sclerosis

Dipeptide repeat proteins activate a heat shock response found in C9ORF72-ALS/FTLD patients

Dipeptide repeat (DPR) pathology in the skeletal muscle of ALS patients with C9ORF72 repeat expansion

CUG initiation and frameshifting enable production of dipeptide repeat proteins from ALS/FTD C9ORF72 transcripts

Repeat length of C9orf72-associated glycine–alanine polypeptides affects their toxicity

Poly-GR dipeptide repeat polymers correlate with neurodegeneration and Clinicopathological subtypes in C9ORF72-related brain disease

Investigating the spread and toxicity of glycine-alanine dipeptides in C9orf72 ALS/FTD using Drosophila melanogaster

Synaptic dysfunction induced by glycine‐alanine dipeptides in C9orf72‐ ALS/FTD is rescued by SV 2 replenishment

Glycine-alanine dipeptide repeats spread rapidly in a repeat length- and age-dependent manner in the fly brain

CRISPR deletion of the C9ORF72 promoter in ALS/FTD patient motor neurons abolishes production of dipeptide repeat proteins and rescues neurodegeneration

Antibodies inhibit transmission and aggregation of C9orf72 poly‐ GA dipeptide repeat proteins

Poly-glycine–alanine exacerbates C9orf72 repeat expansion-mediated DNA damage via sequestration of phosphorylated ATM and loss of nuclear hnRNPA3

Hochschulschrift

Characterization of the C9orf72 dipeptide repeat proteins in frontotemporal dementia and amyotrophic lateral sclerosis

Dipeptide repeat proteins activate a heat shock response found in C9ORF72-ALS/FTLD patients

Dipeptide repeat (DPR) pathology in the skeletal muscle of ALS patients with C9ORF72 repeat expansion

CUG initiation and frameshifting enable production of dipeptide repeat proteins from ALS/FTD C9ORF72 transcripts

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Glycine-alanine dipeptide repeat protein contributes to toxicity in a zebrafish model of C9orf72 associated neurodegeneration

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Other Objects (12)

Repeat length of C9orf72-associated glycine–alanine polypeptides affects their toxicity

Poly-GR dipeptide repeat polymers correlate with neurodegeneration and Clinicopathological subtypes in C9ORF72-related brain disease

Investigating the spread and toxicity of glycine-alanine dipeptides in C9orf72 ALS/FTD using Drosophila melanogaster

Synaptic dysfunction induced by glycine‐alanine dipeptides in C9orf72‐ ALS/FTD is rescued by SV 2 replenishment

Glycine-alanine dipeptide repeats spread rapidly in a repeat length- and age-dependent manner in the fly brain

CRISPR deletion of the C9ORF72 promoter in ALS/FTD patient motor neurons abolishes production of dipeptide repeat proteins and rescues neurodegeneration

Antibodies inhibit transmission and aggregation of C9orf72 poly‐ GA dipeptide repeat proteins

Poly-glycine–alanine exacerbates C9orf72 repeat expansion-mediated DNA damage via sequestration of phosphorylated ATM and loss of nuclear hnRNPA3

Characterization of the C9orf72 dipeptide repeat proteins in frontotemporal dementia and amyotrophic lateral sclerosis

Dipeptide repeat proteins activate a heat shock response found in C9ORF72-ALS/FTLD patients

Dipeptide repeat (DPR) pathology in the skeletal muscle of ALS patients with C9ORF72 repeat expansion

CUG initiation and frameshifting enable production of dipeptide repeat proteins from ALS/FTD C9ORF72 transcripts

Repeat length of C9orf72-associated glycine–alanine polypeptides affects their toxicity

Poly-GR dipeptide repeat polymers correlate with neurodegeneration and Clinicopathological subtypes in C9ORF72-related brain disease

Investigating the spread and toxicity of glycine-alanine dipeptides in C9orf72 ALS/FTD using Drosophila melanogaster

Synaptic dysfunction induced by glycine‐alanine dipeptides in C9orf72‐ ALS/FTD is rescued by SV 2 replenishment

Glycine-alanine dipeptide repeats spread rapidly in a repeat length- and age-dependent manner in the fly brain

CRISPR deletion of the C9ORF72 promoter in ALS/FTD patient motor neurons abolishes production of dipeptide repeat proteins and rescues neurodegeneration

Antibodies inhibit transmission and aggregation of C9orf72 poly‐ GA dipeptide repeat proteins

Poly-glycine–alanine exacerbates C9orf72 repeat expansion-mediated DNA damage via sequestration of phosphorylated ATM and loss of nuclear hnRNPA3

Characterization of the C9orf72 dipeptide repeat proteins in frontotemporal dementia and amyotrophic lateral sclerosis

Dipeptide repeat proteins activate a heat shock response found in C9ORF72-ALS/FTLD patients

Dipeptide repeat (DPR) pathology in the skeletal muscle of ALS patients with C9ORF72 repeat expansion

CUG initiation and frameshifting enable production of dipeptide repeat proteins from ALS/FTD C9ORF72 transcripts

Repeat length of C9orf72-associated glycine–alanine polypeptides affects their toxicity

Poly-GR dipeptide repeat polymers correlate with neurodegeneration and Clinicopathological subtypes in C9ORF72-related brain disease

Investigating the spread and toxicity of glycine-alanine dipeptides in C9orf72 ALS/FTD using Drosophila melanogaster

Synaptic dysfunction induced by glycine‐alanine dipeptides in C9orf72‐ ALS/FTD is rescued by SV 2 replenishment

Glycine-alanine dipeptide repeats spread rapidly in a repeat length- and age-dependent manner in the fly brain

CRISPR deletion of the C9ORF72 promoter in ALS/FTD patient motor neurons abolishes production of dipeptide repeat proteins and rescues neurodegeneration

Antibodies inhibit transmission and aggregation of C9orf72 poly‐ GA dipeptide repeat proteins

Poly-glycine–alanine exacerbates C9orf72 repeat expansion-mediated DNA damage via sequestration of phosphorylated ATM and loss of nuclear hnRNPA3

Characterization of the C9orf72 dipeptide repeat proteins in frontotemporal dementia and amyotrophic lateral sclerosis

Dipeptide repeat proteins activate a heat shock response found in C9ORF72-ALS/FTLD patients

Dipeptide repeat (DPR) pathology in the skeletal muscle of ALS patients with C9ORF72 repeat expansion

CUG initiation and frameshifting enable production of dipeptide repeat proteins from ALS/FTD C9ORF72 transcripts

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