MicroRNA-192 promotes the development of nasopharyngeal carcinoma through targeting RB1 and activating PI3K/AKT pathway

to related objects

Location
Deutsche Nationalbibliothek Frankfurt am Main
ISSN
1477-7819
Extent
Online-Ressource
Language
Englisch
Notes
online resource.

Bibliographic citation
MicroRNA-192 promotes the development of nasopharyngeal carcinoma through targeting RB1 and activating PI3K/AKT pathway ; volume:18 ; number:1 ; day:3 ; month:2 ; year:2020 ; pages:1-9 ; date:12.2020
World journal of surgical oncology ; 18, Heft 1 (3.2.2020), 1-9, 12.2020

Creator
Huang, Qingli
Hou, Sen
Zhu, Xiuqing
Liu, Shouzhou
Contributor
SpringerLink (Online service)

DOI
10.1186/s12957-020-1798-y
URN
urn:nbn:de:101:1-2020050408154389102087
Rights
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
Last update
14.08.2025, 10:58 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Show original at data provider

Associated

  • Huang, Qingli
  • Hou, Sen
  • Zhu, Xiuqing
  • Liu, Shouzhou
  • SpringerLink (Online service)

Other Objects (12)

SETD1A promotes the proliferation and glycolysis of nasopharyngeal carcinoma cells by activating the PI3K/Akt pathway

SETD1A promotes the proliferation and glycolysis of nasopharyngeal carcinoma cells by activating the PI3K/Akt pathway

SOX2 recruits KLF4 to regulate nasopharyngeal carcinoma proliferation via PI3K/AKT signaling

SOX2 recruits KLF4 to regulate nasopharyngeal carcinoma proliferation via PI3K/AKT signaling

SLC25A32 promotes malignant progression of glioblastoma by activating PI3K-AKT signaling pathway

SLC25A32 promotes malignant progression of glioblastoma by activating PI3K-AKT signaling pathway

PENK inhibits osteosarcoma cell migration by activating the PI3K/Akt signaling pathway

PENK inhibits osteosarcoma cell migration by activating the PI3K/Akt signaling pathway

Tideglusib promotes wound healing in aged skin by activating PI3K/Akt pathway

Tideglusib promotes wound healing in aged skin by activating PI3K/Akt pathway

SRSF3 Alleviates Ischemic Cerebral Infarction Damage by Activating the PI3K/AKT Pathway

SRSF3 Alleviates Ischemic Cerebral Infarction Damage by Activating the PI3K/AKT Pathway

Rhein promotes skin wound healing by activating the PI3K/AKT signaling pathway

Rhein promotes skin wound healing by activating the PI3K/AKT signaling pathway

Epigenetic silencing of IGFBPL1 promotes esophageal cancer growth by activating PI3K-AKT signaling

Epigenetic silencing of IGFBPL1 promotes esophageal cancer growth by activating PI3K-AKT signaling

LncRNA DANCR upregulates PI3K/AKT signaling through activating serine phosphorylation of RXRA

LncRNA DANCR upregulates PI3K/AKT signaling through activating serine phosphorylation of RXRA

FLI1 regulates radiotherapy resistance in nasopharyngeal carcinoma through TIE1-mediated PI3K/AKT signaling pathway

FLI1 regulates radiotherapy resistance in nasopharyngeal carcinoma through TIE1-mediated PI3K/AKT signaling pathway

SRC-3/TRAF4 facilitates ovarian cancer development by activating the PI3K/AKT signaling pathway

SRC-3/TRAF4 facilitates ovarian cancer development by activating the PI3K/AKT signaling pathway

USP3 promotes osteosarcoma progression via deubiquitinating EPHA2 and activating the PI3K/AKT signaling pathway

USP3 promotes osteosarcoma progression via deubiquitinating EPHA2 and activating the PI3K/AKT signaling pathway

Related objects