The epithelial-to-mesenchymal transition induced by tumor-associated macrophages confers chemoresistance in peritoneally disseminated pancreatic cancer
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1756-9966
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
The epithelial-to-mesenchymal transition induced by tumor-associated macrophages confers chemoresistance in peritoneally disseminated pancreatic cancer ; volume:37 ; number:1 ; day:11 ; month:12 ; year:2018 ; pages:1-10 ; date:12.2018
Journal of experimental & clinical cancer research ; 37, Heft 1 (11.12.2018), 1-10, 12.2018
- Urheber
-
Kuwada, Kazuya
Kagawa, Shunsuke
Yoshida, Ryuichi
Sakamoto, Shuichi
Ito, Atene
Watanabe, Megumi
Ieda, Takeshi
Kuroda, Shinji
Kikuchi, Satoru
Tazawa, Hiroshi
Fujiwara, Toshiyoshi
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1186/s13046-018-0981-2
- URN
-
urn:nbn:de:101:1-2019012713210070130099
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:50 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Kuwada, Kazuya
- Kagawa, Shunsuke
- Yoshida, Ryuichi
- Sakamoto, Shuichi
- Ito, Atene
- Watanabe, Megumi
- Ieda, Takeshi
- Kuroda, Shinji
- Kikuchi, Satoru
- Tazawa, Hiroshi
- Fujiwara, Toshiyoshi
- SpringerLink (Online service)
Ähnliche Objekte (12)
LMTK3 confers chemo-resistance in breast cancer
Tumor-associated macrophages in meningioma
Tumor‐associated macrophages‐educated reparative macrophages promote diabetic wound healing
Targeting tumor-associated macrophages for cancer treatment
A cytosolic mutp53(E285K) variant confers chemoresistance of malignant melanoma
Crosstalk between tumor-associated macrophages and tumor cells promotes chemoresistance via CXCL5/PI3K/AKT/mTOR pathway in gastric cancer
Microscale Gene Expression Analysis of Tumor-Associated Macrophages
NDRG2 programs tumor-associated macrophages for tumor support
Tumor-Associated Macrophages as Target for Antitumor Therapy
Tumor-associated macrophages, potential targets for cancer treatment
Decoding the spatiotemporal heterogeneity of tumor-associated macrophages
Targeting Squalene Epoxidase Confers Metabolic Vulnerability and Overcomes Chemoresistance in HNSCC
LMTK3 confers chemo-resistance in breast cancer
Tumor-associated macrophages in meningioma
Tumor‐associated macrophages‐educated reparative macrophages promote diabetic wound healing
Targeting tumor-associated macrophages for cancer treatment
A cytosolic mutp53(E285K) variant confers chemoresistance of malignant melanoma
Crosstalk between tumor-associated macrophages and tumor cells promotes chemoresistance via CXCL5/PI3K/AKT/mTOR pathway in gastric cancer
Microscale Gene Expression Analysis of Tumor-Associated Macrophages
NDRG2 programs tumor-associated macrophages for tumor support
Tumor-Associated Macrophages as Target for Antitumor Therapy
Tumor-associated macrophages, potential targets for cancer treatment
Decoding the spatiotemporal heterogeneity of tumor-associated macrophages
Targeting Squalene Epoxidase Confers Metabolic Vulnerability and Overcomes Chemoresistance in HNSCC
LMTK3 confers chemo-resistance in breast cancer
Tumor-associated macrophages in meningioma
Tumor‐associated macrophages‐educated reparative macrophages promote diabetic wound healing
Targeting tumor-associated macrophages for cancer treatment
A cytosolic mutp53(E285K) variant confers chemoresistance of malignant melanoma
Crosstalk between tumor-associated macrophages and tumor cells promotes chemoresistance via CXCL5/PI3K/AKT/mTOR pathway in gastric cancer
Microscale Gene Expression Analysis of Tumor-Associated Macrophages
NDRG2 programs tumor-associated macrophages for tumor support
Tumor-Associated Macrophages as Target for Antitumor Therapy
Tumor-associated macrophages, potential targets for cancer treatment
Decoding the spatiotemporal heterogeneity of tumor-associated macrophages