PSMC5 regulates microglial polarization and activation in LPS-induced cognitive deficits and motor impairments by interacting with TLR4

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: 1 Online-Ressource.

Language: Englisch

Bibliographic citation: PSMC5 regulates microglial polarization and activation in LPS-induced cognitive deficits and motor impairments by interacting with TLR4 ; volume:20 ; number:1 ; day:24 ; month:11 ; year:2023 ; pages:1-20 ; date:12.2023
Journal of neuroinflammation ; 20, Heft 1 (24.11.2023), 1-20, 12.2023

Creator: Bi, Wei
Bao, Keyao
Zhou, Xinqi
Deng, Yihui
Li, Xiaoting
Zhang, Jiawei
Lan, Xin
Zhao, Jiayi
Lu, Daxiang
Xu, Yezi
Cen, Yanmei
Cao, Rui
Xu, Mengyang
Zhong, Wenbin
Zhu, Lihong

Contributor: SpringerLink (Online service)

DOI: 10.1186/s12974-023-02904-9

URN: urn:nbn:de:101:1-2024020714495823072574

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:33 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.

Show original at data provider

Associated

Bi, Wei
Bao, Keyao
Zhou, Xinqi
Deng, Yihui
Li, Xiaoting
Zhang, Jiawei
Lan, Xin
Zhao, Jiayi
Lu, Daxiang
Xu, Yezi
Cen, Yanmei
Cao, Rui
Xu, Mengyang
Zhong, Wenbin
Zhu, Lihong
SpringerLink (Online service)

Other Objects (12)

TLR4 mutation reduces microglial activation, increases Aβ deposits and exacerbates cognitive deficits in a mouse model of Alzheimer's disease

Cascade Signals of Papaverine Inhibiting LPS-Induced Retinal Microglial Activation

Microglial activation by microbial neuraminidase through TLR2 and TLR4 receptors

(+)-JQ1 attenuated LPS-induced microglial inflammation via MAPK/NFκB signaling

Tropomodulin1 exacerbates inflammatory response in macrophages by negatively regulating LPS-induced TLR4 endocytosis

TLR4 and CD14 trafficking and its influence on LPS-induced pro-inflammatory signaling

Flotillins affect LPS-induced TLR4 signaling by modulating the trafficking and abundance of CD14

Role of TLR4-p38 MAPK-Hsp27 signal pathway in LPS-induced pulmonary epithelial hyperpermeability

Effects of ginsenoside Re on LPS-induced inflammatory mediators in BV2 microglial cells

PARK7/DJ-1 deficiency impairs microglial activation in response to LPS-induced inflammation

Correction: Flotillins affect LPS-induced TLR4 signaling by modulating the trafficking and abundance of CD14

Ibrutinib suppresses LPS-induced neuroinflammatory responses in BV2 microglial cells and wild-type mice

TLR4 mutation reduces microglial activation, increases Aβ deposits and exacerbates cognitive deficits in a mouse model of Alzheimer's disease

Cascade Signals of Papaverine Inhibiting LPS-Induced Retinal Microglial Activation

Microglial activation by microbial neuraminidase through TLR2 and TLR4 receptors

(+)-JQ1 attenuated LPS-induced microglial inflammation via MAPK/NFκB signaling

Tropomodulin1 exacerbates inflammatory response in macrophages by negatively regulating LPS-induced TLR4 endocytosis

TLR4 and CD14 trafficking and its influence on LPS-induced pro-inflammatory signaling

Flotillins affect LPS-induced TLR4 signaling by modulating the trafficking and abundance of CD14

Role of TLR4-p38 MAPK-Hsp27 signal pathway in LPS-induced pulmonary epithelial hyperpermeability

Effects of ginsenoside Re on LPS-induced inflammatory mediators in BV2 microglial cells

PARK7/DJ-1 deficiency impairs microglial activation in response to LPS-induced inflammation

Correction: Flotillins affect LPS-induced TLR4 signaling by modulating the trafficking and abundance of CD14

Ibrutinib suppresses LPS-induced neuroinflammatory responses in BV2 microglial cells and wild-type mice

TLR4 mutation reduces microglial activation, increases Aβ deposits and exacerbates cognitive deficits in a mouse model of Alzheimer's disease

Cascade Signals of Papaverine Inhibiting LPS-Induced Retinal Microglial Activation

Microglial activation by microbial neuraminidase through TLR2 and TLR4 receptors

(+)-JQ1 attenuated LPS-induced microglial inflammation via MAPK/NFκB signaling

Tropomodulin1 exacerbates inflammatory response in macrophages by negatively regulating LPS-induced TLR4 endocytosis

TLR4 and CD14 trafficking and its influence on LPS-induced pro-inflammatory signaling

Flotillins affect LPS-induced TLR4 signaling by modulating the trafficking and abundance of CD14

Role of TLR4-p38 MAPK-Hsp27 signal pathway in LPS-induced pulmonary epithelial hyperpermeability

Effects of ginsenoside Re on LPS-induced inflammatory mediators in BV2 microglial cells

PARK7/DJ-1 deficiency impairs microglial activation in response to LPS-induced inflammation

Correction: Flotillins affect LPS-induced TLR4 signaling by modulating the trafficking and abundance of CD14

Ibrutinib suppresses LPS-induced neuroinflammatory responses in BV2 microglial cells and wild-type mice

Cultural heritage institutions wishing to register will find more information here.

Fields marked * need to be filled in.

Username*

Please enter your username

Email*

Please enter your email address

Please do not fill this field

First name

Last name

Password*

Please enter your password

Confirm password*

Please enter the same password

I have read the terms of use and the privacy policy for the collection of personal data and accept them. *

This field is required.

I would like to subscribe to the newsletter of the Deutsche Digitale Bibliothek. See newsletter subscription info.

Account created

Your "My DDB" account has been successfully created. Before you can log in to your account, you must click the confirmation link in the message we just sent to the email address you provided.

PSMC5 regulates microglial polarization and activation in LPS-induced cognitive deficits and motor impairments by interacting with TLR4

Object Details

References and Relationships

Contributors, Places and Time

Further information

Data provider

Associated

Other Objects (12)

TLR4 mutation reduces microglial activation, increases Aβ deposits and exacerbates cognitive deficits in a mouse model of Alzheimer's disease

Cascade Signals of Papaverine Inhibiting LPS-Induced Retinal Microglial Activation

Microglial activation by microbial neuraminidase through TLR2 and TLR4 receptors

(+)-JQ1 attenuated LPS-induced microglial inflammation via MAPK/NFκB signaling

Tropomodulin1 exacerbates inflammatory response in macrophages by negatively regulating LPS-induced TLR4 endocytosis

TLR4 and CD14 trafficking and its influence on LPS-induced pro-inflammatory signaling

Flotillins affect LPS-induced TLR4 signaling by modulating the trafficking and abundance of CD14

Role of TLR4-p38 MAPK-Hsp27 signal pathway in LPS-induced pulmonary epithelial hyperpermeability

Effects of ginsenoside Re on LPS-induced inflammatory mediators in BV2 microglial cells

PARK7/DJ-1 deficiency impairs microglial activation in response to LPS-induced inflammation

Correction: Flotillins affect LPS-induced TLR4 signaling by modulating the trafficking and abundance of CD14

Ibrutinib suppresses LPS-induced neuroinflammatory responses in BV2 microglial cells and wild-type mice

TLR4 mutation reduces microglial activation, increases Aβ deposits and exacerbates cognitive deficits in a mouse model of Alzheimer's disease

Cascade Signals of Papaverine Inhibiting LPS-Induced Retinal Microglial Activation

Microglial activation by microbial neuraminidase through TLR2 and TLR4 receptors

(+)-JQ1 attenuated LPS-induced microglial inflammation via MAPK/NFκB signaling

Tropomodulin1 exacerbates inflammatory response in macrophages by negatively regulating LPS-induced TLR4 endocytosis

TLR4 and CD14 trafficking and its influence on LPS-induced pro-inflammatory signaling

Flotillins affect LPS-induced TLR4 signaling by modulating the trafficking and abundance of CD14

Role of TLR4-p38 MAPK-Hsp27 signal pathway in LPS-induced pulmonary epithelial hyperpermeability

Effects of ginsenoside Re on LPS-induced inflammatory mediators in BV2 microglial cells

PARK7/DJ-1 deficiency impairs microglial activation in response to LPS-induced inflammation

Correction: Flotillins affect LPS-induced TLR4 signaling by modulating the trafficking and abundance of CD14

Ibrutinib suppresses LPS-induced neuroinflammatory responses in BV2 microglial cells and wild-type mice

TLR4 mutation reduces microglial activation, increases Aβ deposits and exacerbates cognitive deficits in a mouse model of Alzheimer's disease

Cascade Signals of Papaverine Inhibiting LPS-Induced Retinal Microglial Activation

Microglial activation by microbial neuraminidase through TLR2 and TLR4 receptors

(+)-JQ1 attenuated LPS-induced microglial inflammation via MAPK/NFκB signaling

Tropomodulin1 exacerbates inflammatory response in macrophages by negatively regulating LPS-induced TLR4 endocytosis

TLR4 and CD14 trafficking and its influence on LPS-induced pro-inflammatory signaling

Flotillins affect LPS-induced TLR4 signaling by modulating the trafficking and abundance of CD14

Role of TLR4-p38 MAPK-Hsp27 signal pathway in LPS-induced pulmonary epithelial hyperpermeability

Effects of ginsenoside Re on LPS-induced inflammatory mediators in BV2 microglial cells

PARK7/DJ-1 deficiency impairs microglial activation in response to LPS-induced inflammation

Correction: Flotillins affect LPS-induced TLR4 signaling by modulating the trafficking and abundance of CD14

Ibrutinib suppresses LPS-induced neuroinflammatory responses in BV2 microglial cells and wild-type mice

Related objects

Reset password