Double Ventricular Response with Aberrant Conduction Leading to Ventricular Dysfunction
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
1 Online-Ressource.
- Language
-
Englisch
- Bibliographic citation
-
Double Ventricular Response with Aberrant Conduction Leading to Ventricular Dysfunction ; day:6 ; month:5 ; year:2024 ; pages:1-5
Pediatric cardiology ; (6.5.2024), 1-5
- Creator
-
Warren, Paul W.
Powell, Adam W.
Knilans, Timothy
Connor, Chad
Baskar, Shankar
- Contributor
-
SpringerLink (Online service)
- DOI
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10.1007/s00246-024-03506-9
- URN
-
urn:nbn:de:101:1-2407152100296.501994572995
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 11:01 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Warren, Paul W.
- Powell, Adam W.
- Knilans, Timothy
- Connor, Chad
- Baskar, Shankar
- SpringerLink (Online service)
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Right Ventricular Dysfunction contributes to Left Ventricular Thrombus Formation in Patients post Anterior Myocardial Infarction
Recent advances in ventricular conduction
Left ventricular dysfunction
Association of peripheral nerve conduction in diabetic neuropathy with subclinical left ventricular systolic dysfunction
Aortic stenosis and right ventricular dysfunction
Right ventricular dysfunction in Chagas' Cardiomyopathy. Primary involvement or a resonant manifestation of Left ventricular dysfunction?
A novel echocardiographic right ventricular dysfunction score can identify hemodynamic severity profiles in left ventricular dysfunction
Left ventricular geometry predicts ventricular tachyarrhythmia in patients with left ventricular systolic dysfunction: a comprehensive cardiovascular magnetic resonance study
4D flow CMR can detect subtle right ventricular dysfunction in primary left ventricular disease
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HDAC5 inhibition attenuates ventricular remodeling and cardiac dysfunction
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