ASC- and caspase-8-dependent apoptotic pathway diverges from the NLRC4 inflammasome in macrophages
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2045-2322
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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ASC- and caspase-8-dependent apoptotic pathway diverges from the NLRC4 inflammasome in macrophages ; volume:8 ; number:1 ; day:28 ; month:2 ; year:2018 ; pages:1-12 ; date:12.2018
Scientific reports ; 8, Heft 1 (28.2.2018), 1-12, 12.2018
- Creator
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Lee, Bettina L.
- Contributor
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Mirrashidi, Kathleen M.
Stowe, Irma B.
Kummerfeld, Sarah K.
Watanabe, Colin
Haley, Benjamin
Cuellar, Trinna L.
Reichelt, Michael
Kayagaki, Nobuhiko
SpringerLink (Online service)
- DOI
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10.1038/s41598-018-21998-3
- URN
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urn:nbn:de:101:1-2018091022484851282920
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:21 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Lee, Bettina L.
- Mirrashidi, Kathleen M.
- Stowe, Irma B.
- Kummerfeld, Sarah K.
- Watanabe, Colin
- Haley, Benjamin
- Cuellar, Trinna L.
- Reichelt, Michael
- Kayagaki, Nobuhiko
- SpringerLink (Online service)
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Caspase-11 mediated inflammasome activation in macrophages by systemic infection of A. actinomycetemcomitans exacerbates arthritis
Directly targeting ASC by lonidamine alleviates inflammasome-driven diseases
ASC nanobodies to counteract the consequences of inflammasome activation
Apoptotic signaling beyond caspase activation in hepatocytes
Activation of the NLRP1B inflammasome by caspase-8
Inflammasome activity is controlled by ZBTB16-dependent SUMOylation of ASC
ASC Speck Formation after Inflammasome Activation in Primary Human Keratinocytes
ASC specks exacerbate α‑synuclein pathology via amplifying NLRP3 inflammasome activities
Apoptotic cells induce proliferation of peritoneal macrophages
The role of Caspase-8 in regulating apoptotic and non-apoptotic pathways is gynecological cancers
The NLRP3-inflammasome-caspase-1 pathway is upregulated in idiopathic pulmonary fibrosis and acute exacerbations and is inducible by apoptotic A549 cells
AIP1 Regulates Ocular Angiogenesis Via NLRP12‐ASC‐Caspase‐8 Inflammasome‐Mediated Endothelial Pyroptosis
Caspase-11 mediated inflammasome activation in macrophages by systemic infection of A. actinomycetemcomitans exacerbates arthritis
Directly targeting ASC by lonidamine alleviates inflammasome-driven diseases
ASC nanobodies to counteract the consequences of inflammasome activation
Apoptotic signaling beyond caspase activation in hepatocytes
Activation of the NLRP1B inflammasome by caspase-8
Inflammasome activity is controlled by ZBTB16-dependent SUMOylation of ASC
ASC Speck Formation after Inflammasome Activation in Primary Human Keratinocytes
ASC specks exacerbate α‑synuclein pathology via amplifying NLRP3 inflammasome activities
Apoptotic cells induce proliferation of peritoneal macrophages
The role of Caspase-8 in regulating apoptotic and non-apoptotic pathways is gynecological cancers