Hochschulschrift
A high-complexity lentiviral shRNA screen identifies synthetic lethal interactions with deregulated N-Myc in neuroblastoma cells
- Alternative title
-
Ein hoch-Komplexität Genom-weit RNAi Screen für synthetisch letale Interaktion mit dereguliertem N-Myc in Neuroblastomzellen
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
Würzburg, Universität Würzburg, Dissertation, 2014
- Keyword
-
Neuroblastom
N-Myc
Deregulierung
RNS-Interferenz
Krebs
Onkologie
- Event
-
Veröffentlichung
- (where)
-
Würzburg
- (who)
-
Universität Würzburg
- (when)
-
2014
- Creator
- Contributor
-
Eilers, Martin
- URN
-
urn:nbn:de:bvb:20-opus-103157
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:53 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Object type
- Hochschulschrift
Associated
- Xu, Jiajia
- Eilers, Martin
- Universität Würzburg
Time of origin
- 2014
Other Objects (12)
A high-complexity lentiviral shRNA screen identifies synthetic lethal interactions with deregulated N-Myc in neuroblastoma cells
Decoding the metabolic program of deregulated MYCN expression in neuroblastoma
A Selective Small‐Molecule c‐Myc Degrader Potently Regresses Lethal c‐Myc Overexpressing Tumors
Deregulated expression of microRNAs : a critical pathogenic mechanism in MYC-induced lymphoma
Mitoribosomal synthetic lethality overcomes multidrug resistance in MYC-driven neuroblastoma
N-Myc-induced metabolic rewiring creates novel therapeutic vulnerabilities in neuroblastoma
Extracellular matrix rigidity modulates neuroblastoma cell differentiation and N-myc expression
MYC and the unfolded protein response in cancer: synthetic lethal partners in crime?
The lethal giant larvaetumour suppressor mutation requires dMyc oncoprotein to promote clonal malignancy
Genetic screening for anticancer genes highlights FBLN5 as a synthetic lethal partner of MYC
The initial mechanism involved in N-myc oncogene amplification in human neuroblastoma cell lines
Transcription factor activating protein 4 is synthetically lethal and a master regulator of MYCN-amplified neuroblastoma
A high-complexity lentiviral shRNA screen identifies synthetic lethal interactions with deregulated N-Myc in neuroblastoma cells
Decoding the metabolic program of deregulated MYCN expression in neuroblastoma
A Selective Small‐Molecule c‐Myc Degrader Potently Regresses Lethal c‐Myc Overexpressing Tumors
Deregulated expression of microRNAs : a critical pathogenic mechanism in MYC-induced lymphoma
Mitoribosomal synthetic lethality overcomes multidrug resistance in MYC-driven neuroblastoma
N-Myc-induced metabolic rewiring creates novel therapeutic vulnerabilities in neuroblastoma
Extracellular matrix rigidity modulates neuroblastoma cell differentiation and N-myc expression
MYC and the unfolded protein response in cancer: synthetic lethal partners in crime?
The lethal giant larvaetumour suppressor mutation requires dMyc oncoprotein to promote clonal malignancy
Genetic screening for anticancer genes highlights FBLN5 as a synthetic lethal partner of MYC
The initial mechanism involved in N-myc oncogene amplification in human neuroblastoma cell lines
Transcription factor activating protein 4 is synthetically lethal and a master regulator of MYCN-amplified neuroblastoma
A high-complexity lentiviral shRNA screen identifies synthetic lethal interactions with deregulated N-Myc in neuroblastoma cells
Decoding the metabolic program of deregulated MYCN expression in neuroblastoma
A Selective Small‐Molecule c‐Myc Degrader Potently Regresses Lethal c‐Myc Overexpressing Tumors
Deregulated expression of microRNAs : a critical pathogenic mechanism in MYC-induced lymphoma
Mitoribosomal synthetic lethality overcomes multidrug resistance in MYC-driven neuroblastoma
N-Myc-induced metabolic rewiring creates novel therapeutic vulnerabilities in neuroblastoma
Extracellular matrix rigidity modulates neuroblastoma cell differentiation and N-myc expression
MYC and the unfolded protein response in cancer: synthetic lethal partners in crime?
The lethal giant larvaetumour suppressor mutation requires dMyc oncoprotein to promote clonal malignancy
Genetic screening for anticancer genes highlights FBLN5 as a synthetic lethal partner of MYC
The initial mechanism involved in N-myc oncogene amplification in human neuroblastoma cell lines