Correction to: Disruption of endothelial adherens junctions by high glucose is mediated by protein kinase C-β–dependent vascular endothelial cadherin tyrosine phosphorylation
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1475-2840
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Correction to: Disruption of endothelial adherens junctions by high glucose is mediated by protein kinase C-β–dependent vascular endothelial cadherin tyrosine phosphorylation ; volume:16 ; number:1 ; day:20 ; month:10 ; year:2017 ; pages:1 ; date:12.2017
Cardiovascular diabetology ; 16, Heft 1 (20.10.2017), 1, 12.2017
- Keyword
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Kardiovaskuläre Krankheit
- Creator
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Haidari, Mehran
- Contributor
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Zhang, Wei
Willerson, James T.
Dixon, Richard A. F.
SpringerLink (Online service)
- DOI
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10.1186/s12933-017-0614-7
- URN
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urn:nbn:de:1111-201712151954
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:34 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Haidari, Mehran
- Zhang, Wei
- Willerson, James T.
- Dixon, Richard A. F.
- SpringerLink (Online service)
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Kaposi's sarcoma-associated herpes virus disrupts adherens junctions and increases endothelial permeability by inducing degradation of VE-cadherin
Drebrin preserves endothelial integrity by stabilizing nectin at adherens junctions
ESAM controls opening of endothelial junctions via phosphorylation of VE-cadherin at Y685 residue
Ubiquitin ligase CHFR mediated degradation of VE-cadherin through ubiquitylation disrupts endothelial adherens junctions
Vascular endothelial cadherin is an endostatin receptor
Interplay between neural-cadherin and vascular endothelial-cadherin in breast cancer progression
Histamine causes endothelial barrier disruption via Ca2+-mediated RhoA activation and tension at adherens junctions
CU06-1004 (endothelial dysfunction blocker) ameliorates astrocyte end-feet swelling by stabilizing endothelial cell junctions in cerebral ischemia/reperfusion injury
AGE-BSA down-regulates endothelial connexin43 gap junctions
The roles of VE-cadherin-serine 665 and -tyrosine 685 for endocytosis in the control of endothelial junctions
CMTM4 regulates angiogenesis by promoting cell surface recycling of VE-cadherin to endothelial adherens junctions
Pasteurella multocida infection induces blood–brain barrier disruption by decreasing tight junctions and adherens junctions between neighbored brain microvascular endothelial cells
Kaposi's sarcoma-associated herpes virus disrupts adherens junctions and increases endothelial permeability by inducing degradation of VE-cadherin
Drebrin preserves endothelial integrity by stabilizing nectin at adherens junctions
ESAM controls opening of endothelial junctions via phosphorylation of VE-cadherin at Y685 residue
Ubiquitin ligase CHFR mediated degradation of VE-cadherin through ubiquitylation disrupts endothelial adherens junctions
Vascular endothelial cadherin is an endostatin receptor
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Histamine causes endothelial barrier disruption via Ca2+-mediated RhoA activation and tension at adherens junctions
CU06-1004 (endothelial dysfunction blocker) ameliorates astrocyte end-feet swelling by stabilizing endothelial cell junctions in cerebral ischemia/reperfusion injury
AGE-BSA down-regulates endothelial connexin43 gap junctions