Dopaminergic neuron loss in mice due to increased levels of wild-type human α-Synuclein only takes place under conditions of accelerated aging
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Dopaminergic neuron loss in mice due to increased levels of wild-type human α-Synuclein only takes place under conditions of accelerated aging ; volume:14 ; number:1 ; day:30 ; month:1 ; year:2024 ; pages:1-15 ; date:12.2024
Scientific reports ; 14, Heft 1 (30.1.2024), 1-15, 12.2024
- Creator
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Perez-Villalba, Ana
Sirerol-Piquer, María Salomé
Soriano-Cantón, Raúl
Folgado, Virginia
Pérez-Cañamás, Azucena
Kirstein, Martina
Fariñas, Isabel
Pérez-Sánchez, Francisco
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41598-024-53093-1
- URN
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urn:nbn:de:101:1-2024041011073130608628
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 11:00 AM CEST
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Associated
- Perez-Villalba, Ana
- Sirerol-Piquer, María Salomé
- Soriano-Cantón, Raúl
- Folgado, Virginia
- Pérez-Cañamás, Azucena
- Kirstein, Martina
- Fariñas, Isabel
- Pérez-Sánchez, Francisco
- SpringerLink (Online service)
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α-Synuclein fibril-specific nanobody reduces prion-like α-synuclein spreading in mice
Functionalization of α-synuclein fibrils
Early-onset impairment of the ubiquitin-proteasome system in dopaminergic neurons caused by α-synuclein
Protection from α-Synuclein induced dopaminergic neurodegeneration by overexpression of the mitochondrial import receptor TOM20
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27-Hydroxycholesterol increases α-synuclein protein levels through proteasomal inhibition in human dopaminergic neurons
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Protection from α-Synuclein induced dopaminergic neurodegeneration by overexpression of the mitochondrial import receptor TOM20
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