TLR4-mediated IL-12 production enhances IFN-γ and IL-1β production, which inhibits TGF-β production and promotes antibody-induced joint inflammation

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1478-6354

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: TLR4-mediated IL-12 production enhances IFN-γ and IL-1β production, which inhibits TGF-β production and promotes antibody-induced joint inflammation ; volume:14 ; number:5 ; day:4 ; month:10 ; year:2012 ; pages:1-13 ; date:10.2012
Arthritis Research & Therapy ; 14, Heft 5 (4.10.2012), 1-13, 10.2012

Creator: Kim, Hye Sung

Contributor: Chung, Doo Hyun
SpringerLink (Online service)

DOI: 10.1186/ar4048

URN: urn:nbn:de:1111-2016050323657

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 11:01 AM CEST

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Kim, Hye Sung
Chung, Doo Hyun
SpringerLink (Online service)

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Regulation of IL-18 binding protein by IFN-γ [IFN-gamma]

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Regulation of IL-18 binding protein by IFN-γ [IFN-gamma]

Regulation of IFN‐γ production by ZFP36L2 in T cells is time‐dependent

Erratum to: Leishmania infantum-specific production of IFN-γ and IL-10 in stimulated blood from dogs with clinical leishmaniosis

Methylprednisolone inhibits IFN-γ and IL-17 expression and production by cells infiltrating central nervous system in experimental autoimmune encephalomyelitis

Human genetic GLUT1 deficiency results in impaired T cellular IFN-γ production : = Human genetic GLUT1 deficiency results in impaired T cellular IFN-[gamma] production

C5aR1 activation drives early IFN-γ production to control experimental toxoplasma gondii infection

Human T cells employ conserved AU‐rich elements to fine‐tune IFN‐γ production

IL-18 associated with lung lymphoid aggregates drives IFNγ production in severe COPD

IFNG +874T/A polymorphism is not associated with American tegumentary leishmaniasis susceptibility but can influence Leishmaniainduced IFN-γ production

Leishmania infantum-specific production of IFN-γ and IL-10 in stimulated blood from dogs with clinical leishmaniosis

Human Genetic GLUT1 Deficiency Results in Impaired T Cellular IFN‐γ Production

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Regulation of IL-18 binding protein by IFN-γ [IFN-gamma]

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Regulation of IL-18 binding protein by IFN-γ [IFN-gamma]

Regulation of IFN‐γ production by ZFP36L2 in T cells is time‐dependent

Erratum to: Leishmania infantum-specific production of IFN-γ and IL-10 in stimulated blood from dogs with clinical leishmaniosis

Methylprednisolone inhibits IFN-γ and IL-17 expression and production by cells infiltrating central nervous system in experimental autoimmune encephalomyelitis

Human genetic GLUT1 deficiency results in impaired T cellular IFN-γ production : = Human genetic GLUT1 deficiency results in impaired T cellular IFN-[gamma] production

C5aR1 activation drives early IFN-γ production to control experimental toxoplasma gondii infection

Human T cells employ conserved AU‐rich elements to fine‐tune IFN‐γ production

IL-18 associated with lung lymphoid aggregates drives IFNγ production in severe COPD

IFNG +874T/A polymorphism is not associated with American tegumentary leishmaniasis susceptibility but can influence Leishmaniainduced IFN-γ production

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TLR4-mediated IL-12 production enhances IFN-γ and IL-1β production, which inhibits TGF-β production and promotes antibody-induced joint inflammation

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Other Objects (12)

Leishmania infantum-specific production of IFN-γ and IL-10 in stimulated blood from dogs with clinical leishmaniosis

Human Genetic GLUT1 Deficiency Results in Impaired T Cellular IFN‐γ Production

Regulation of IL-18 binding protein by IFN-γ [IFN-gamma]

Regulation of IL-18 binding protein by IFN-γ [IFN-gamma]

Regulation of IFN‐γ production by ZFP36L2 in T cells is time‐dependent

Erratum to: Leishmania infantum-specific production of IFN-γ and IL-10 in stimulated blood from dogs with clinical leishmaniosis

Methylprednisolone inhibits IFN-γ and IL-17 expression and production by cells infiltrating central nervous system in experimental autoimmune encephalomyelitis

Human genetic GLUT1 deficiency results in impaired T cellular IFN-γ production : = Human genetic GLUT1 deficiency results in impaired T cellular IFN-[gamma] production

C5aR1 activation drives early IFN-γ production to control experimental toxoplasma gondii infection

Human T cells employ conserved AU‐rich elements to fine‐tune IFN‐γ production

IL-18 associated with lung lymphoid aggregates drives IFNγ production in severe COPD

IFNG +874T/A polymorphism is not associated with American tegumentary leishmaniasis susceptibility but can influence Leishmaniainduced IFN-γ production

Leishmania infantum-specific production of IFN-γ and IL-10 in stimulated blood from dogs with clinical leishmaniosis

Human Genetic GLUT1 Deficiency Results in Impaired T Cellular IFN‐γ Production

Regulation of IL-18 binding protein by IFN-γ [IFN-gamma]

Regulation of IL-18 binding protein by IFN-γ [IFN-gamma]

Regulation of IFN‐γ production by ZFP36L2 in T cells is time‐dependent

Erratum to: Leishmania infantum-specific production of IFN-γ and IL-10 in stimulated blood from dogs with clinical leishmaniosis

Methylprednisolone inhibits IFN-γ and IL-17 expression and production by cells infiltrating central nervous system in experimental autoimmune encephalomyelitis

Human genetic GLUT1 deficiency results in impaired T cellular IFN-γ production : = Human genetic GLUT1 deficiency results in impaired T cellular IFN-[gamma] production

C5aR1 activation drives early IFN-γ production to control experimental toxoplasma gondii infection

Human T cells employ conserved AU‐rich elements to fine‐tune IFN‐γ production

IL-18 associated with lung lymphoid aggregates drives IFNγ production in severe COPD

IFNG +874T/A polymorphism is not associated with American tegumentary leishmaniasis susceptibility but can influence Leishmaniainduced IFN-γ production

Leishmania infantum-specific production of IFN-γ and IL-10 in stimulated blood from dogs with clinical leishmaniosis

Human Genetic GLUT1 Deficiency Results in Impaired T Cellular IFN‐γ Production

Regulation of IL-18 binding protein by IFN-γ [IFN-gamma]

Regulation of IL-18 binding protein by IFN-γ [IFN-gamma]

Regulation of IFN‐γ production by ZFP36L2 in T cells is time‐dependent

Erratum to: Leishmania infantum-specific production of IFN-γ and IL-10 in stimulated blood from dogs with clinical leishmaniosis

Methylprednisolone inhibits IFN-γ and IL-17 expression and production by cells infiltrating central nervous system in experimental autoimmune encephalomyelitis

Human genetic GLUT1 deficiency results in impaired T cellular IFN-γ production : = Human genetic GLUT1 deficiency results in impaired T cellular IFN-[gamma] production

C5aR1 activation drives early IFN-γ production to control experimental toxoplasma gondii infection

Human T cells employ conserved AU‐rich elements to fine‐tune IFN‐γ production

IL-18 associated with lung lymphoid aggregates drives IFNγ production in severe COPD

IFNG +874T/A polymorphism is not associated with American tegumentary leishmaniasis susceptibility but can influence Leishmaniainduced IFN-γ production

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