- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1423-0127
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
The miR-15b-Smurf2-HSP27 axis promotes pulmonary fibrosis ; volume:30 ; number:1 ; day:7 ; month:1 ; year:2023 ; pages:1-15 ; date:12.2023
Journal of biomedical science ; 30, Heft 1 (7.1.2023), 1-15, 12.2023
- Urheber
-
Jeon, Seulgi
Jin, Hee
Kim, Jin-Mo
Hur, Youmin
Song, Eun Joo
Lee, Yoon-Jin
Na, Younghwa
Cho, Jaeho
Lee, Yun-Sil
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1186/s12929-023-00896-5
- URN
-
urn:nbn:de:101:1-2023031620593426842897
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:44 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Jeon, Seulgi
- Jin, Hee
- Kim, Jin-Mo
- Hur, Youmin
- Song, Eun Joo
- Lee, Yoon-Jin
- Na, Younghwa
- Cho, Jaeho
- Lee, Yun-Sil
- SpringerLink (Online service)
Ähnliche Objekte (12)
BDNF/TrkB axis activation promotes epithelial–mesenchymal transition in idiopathic pulmonary fibrosis
Succinate promotes pulmonary fibrosis through GPR91 and predicts death in idiopathic pulmonary fibrosis
The sialidase NEU3 promotes pulmonary fibrosis in mice
Histological hallmarks and role of Slug/PIP axis in pulmonary hypertension secondary to pulmonary fibrosis
ErbB4 promotes M2 activation of macrophages in idiopathic pulmonary fibrosis
P2Y6 Receptor Activation Promotes Inflammation and Tissue Remodeling in Pulmonary Fibrosis
Idiopathic pulmonary fibrosis
Idiopathic Pulmonary Fibrosis
Pulmonary hypertension in idiopathic pulmonary fibrosis
SULF1 expression is increased and promotes fibrosis through the TGF-β1/SMAD pathway in idiopathic pulmonary fibrosis
LncRNA MEG8 promotes NSCLC progression by modulating the miR-15a-5p-miR-15b-5p/PSAT1 axis
15b
BDNF/TrkB axis activation promotes epithelial–mesenchymal transition in idiopathic pulmonary fibrosis
Succinate promotes pulmonary fibrosis through GPR91 and predicts death in idiopathic pulmonary fibrosis
The sialidase NEU3 promotes pulmonary fibrosis in mice
Histological hallmarks and role of Slug/PIP axis in pulmonary hypertension secondary to pulmonary fibrosis
ErbB4 promotes M2 activation of macrophages in idiopathic pulmonary fibrosis
P2Y6 Receptor Activation Promotes Inflammation and Tissue Remodeling in Pulmonary Fibrosis
Idiopathic pulmonary fibrosis
Idiopathic Pulmonary Fibrosis
Pulmonary hypertension in idiopathic pulmonary fibrosis
SULF1 expression is increased and promotes fibrosis through the TGF-β1/SMAD pathway in idiopathic pulmonary fibrosis
LncRNA MEG8 promotes NSCLC progression by modulating the miR-15a-5p-miR-15b-5p/PSAT1 axis
15b
BDNF/TrkB axis activation promotes epithelial–mesenchymal transition in idiopathic pulmonary fibrosis
Succinate promotes pulmonary fibrosis through GPR91 and predicts death in idiopathic pulmonary fibrosis
The sialidase NEU3 promotes pulmonary fibrosis in mice
Histological hallmarks and role of Slug/PIP axis in pulmonary hypertension secondary to pulmonary fibrosis
ErbB4 promotes M2 activation of macrophages in idiopathic pulmonary fibrosis
P2Y6 Receptor Activation Promotes Inflammation and Tissue Remodeling in Pulmonary Fibrosis
Idiopathic pulmonary fibrosis
Idiopathic Pulmonary Fibrosis
Pulmonary hypertension in idiopathic pulmonary fibrosis
SULF1 expression is increased and promotes fibrosis through the TGF-β1/SMAD pathway in idiopathic pulmonary fibrosis
LncRNA MEG8 promotes NSCLC progression by modulating the miR-15a-5p-miR-15b-5p/PSAT1 axis