Phenotypes of pain behavior in phospholipase C-related but catalytically inactive protein type 1 knockout mice
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1744-8069
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
-
Phenotypes of pain behavior in phospholipase C-related but catalytically inactive protein type 1 knockout mice ; volume:7 ; number:1 ; day:18 ; month:10 ; year:2011 ; pages:1-8 ; date:12.2011
Molecular pain ; 7, Heft 1 (18.10.2011), 1-8, 12.2011
- Creator
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Migita, Keisuke
Tomiyama, Masahiko
Yamada, Junko
Fukuzawa, Masashi
Kanematsu, Takashi
Hirata, Masato
Ueno, Shinya
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/1744-8069-7-79
- URN
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urn:nbn:de:101:1-2021090121254439129381
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:31 AM CEST
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Associated
- Migita, Keisuke
- Tomiyama, Masahiko
- Yamada, Junko
- Fukuzawa, Masashi
- Kanematsu, Takashi
- Hirata, Masato
- Ueno, Shinya
- SpringerLink (Online service)
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Phospholipase C-related but catalytically inactive protein modulates pain behavior in a neuropathic pain model in mice
Catalytically inactive long prokaryotic Argonaute systems employ distinct effectors to confer immunity via abortive infection
Creating highly specific nucleases by fusion of active restriction endonucleases and catalytically inactive homing endonucleases
Sensing the DNA-mismatch tolerance of catalytically inactive Cas9 via barcoded DNA nanostructures in solid-state nanopores
Abnormal hematopoietic phenotypes in Pim kinase triple knockout mice
Gene silencing based on RNA-guided catalytically inactive Cas9 (dCas9): a new tool for genetic engineering in Leptospira
Author Correction: Sensing the DNA-mismatch tolerance of catalytically inactive Cas9 via barcoded DNA nanostructures in solid-state nanopores
Characterization of Rett Syndrome-like phenotypes in Mecp2-knockout rats
Late development of OCD-like phenotypes in Dlgap1 knockout mice
II. Inactive Mitglieder.
Enhancing structural plasticity of PC12 neurons during differentiation and neurite regeneration with a catalytically inactive mutant version of the zRICH protein
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Phospholipase C-related but catalytically inactive protein modulates pain behavior in a neuropathic pain model in mice
Catalytically inactive long prokaryotic Argonaute systems employ distinct effectors to confer immunity via abortive infection
Creating highly specific nucleases by fusion of active restriction endonucleases and catalytically inactive homing endonucleases
Sensing the DNA-mismatch tolerance of catalytically inactive Cas9 via barcoded DNA nanostructures in solid-state nanopores
Abnormal hematopoietic phenotypes in Pim kinase triple knockout mice
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Author Correction: Sensing the DNA-mismatch tolerance of catalytically inactive Cas9 via barcoded DNA nanostructures in solid-state nanopores
Characterization of Rett Syndrome-like phenotypes in Mecp2-knockout rats
Late development of OCD-like phenotypes in Dlgap1 knockout mice
II. Inactive Mitglieder.
Enhancing structural plasticity of PC12 neurons during differentiation and neurite regeneration with a catalytically inactive mutant version of the zRICH protein