Interface Gain-of-Function Mutations in TLR7 Cause Systemic and Neuro-inflammatory Disease
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Interface Gain-of-Function Mutations in TLR7 Cause Systemic and Neuro-inflammatory Disease ; volume:44 ; number:2 ; day:7 ; month:2 ; year:2024 ; pages:1-9 ; date:2.2024
Journal of clinical immunology ; 44, Heft 2 (7.2.2024), 1-9, 2.2024
- Creator
-
David, Clémence
Badonyi, Mihaly
Kechiche, Robin
Insalaco, Antonella
Zecca, Marco
Benedetti, Fabrizio De
Orcesi, Simona
Chiapparini, Luisa
Comoli, Patrizia
Federici, Silvia
Gattorno, Marco
Ginevrino, Monia
Giorgio, Elisa
Matteo, Valentina
Moran-Alvarez, Patricia
Politano, Davide
Prencipe, Giusi
Sirchia, Fabio
Volpi, Stefano
Masson, Cécile
Rice, Gillian I.
Frémond, Marie-Louise
Lepelley, Alice
Marsh, Joseph A.
Crow, Yanick J.
- Contributor
-
SpringerLink (Online service)
- DOI
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10.1007/s10875-024-01660-6
- URN
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urn:nbn:de:101:1-2404260705563.245877488154
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:57 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- David, Clémence
- Badonyi, Mihaly
- Kechiche, Robin
- Insalaco, Antonella
- Zecca, Marco
- Benedetti, Fabrizio De
- Orcesi, Simona
- Chiapparini, Luisa
- Comoli, Patrizia
- Federici, Silvia
- Gattorno, Marco
- Ginevrino, Monia
- Giorgio, Elisa
- Matteo, Valentina
- Moran-Alvarez, Patricia
- Politano, Davide
- Prencipe, Giusi
- Sirchia, Fabio
- Volpi, Stefano
- Masson, Cécile
- Rice, Gillian I.
- Frémond, Marie-Louise
- Lepelley, Alice
- Marsh, Joseph A.
- Crow, Yanick J.
- SpringerLink (Online service)
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MDA5 gain-of-function associated with a Glu794del mutation
The role of B cells in neuro-inflammatory diseases
PITX2 gain-of-function induced defects in mouse forelimb development
The curious case of Gαs gain-of-function in neoplasia
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