Bis(9)-(−)-Meptazinol, a novel dual-binding AChE inhibitor, rescues cognitive deficits and pathological changes in APP/PS1 transgenic mice

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2047-9158

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Bis(9)-(−)-Meptazinol, a novel dual-binding AChE inhibitor, rescues cognitive deficits and pathological changes in APP/PS1 transgenic mice ; volume:7 ; number:1 ; day:11 ; month:9 ; year:2018 ; pages:1-11 ; date:12.2018
Translational neurodegeneration ; 7, Heft 1 (11.9.2018), 1-11, 12.2018

Creator: Shi, Yuhuan

Contributor: Huang, Wanying
Wang, Yu
Zhang, Rui
Hou, Lina
Xu, Jianrong
Qiu, Zhuibai
Xie, Qiong
Chen, Hongzhuan
Zhang, Yongfang
Wang, Hao
SpringerLink (Online service)

DOI: 10.1186/s40035-018-0126-8

URN: urn:nbn:de:101:1-2018110315451246342778

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:52 AM CEST

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Associated

Shi, Yuhuan
Huang, Wanying
Wang, Yu
Zhang, Rui
Hou, Lina
Xu, Jianrong
Qiu, Zhuibai
Xie, Qiong
Chen, Hongzhuan
Zhang, Yongfang
Wang, Hao
SpringerLink (Online service)

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Bis(9)-(−)-Meptazinol, a novel dual-binding AChE inhibitor, rescues cognitive deficits and pathological changes in APP/PS1 transgenic mice

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Other Objects (12)

Influenza vaccination in early Alzheimer’s disease rescues amyloidosis and ameliorates cognitive deficits in APP/PS1 mice by inhibiting regulatory T cells

Inhibition of DYRK1A proteolysis modifies its kinase specificity and rescues Alzheimer phenotype in APP/PS1 mice

Pan-PPAR Modulation Effectively Protects APP/PS1 Mice from Amyloid Deposition and Cognitive Deficits

Immune senescence in aged APP/PS1 mice

Deletion of aquaporin-4 in APP/PS1 mice exacerbates brain Aβ accumulation and memory deficits

Rescue of cognitive deficits in APP/PS1 mice by accelerating the aggregation of β-amyloid peptide

Minor ginsenoside F1 improves memory in APP/PS1 mice

Active constituent of Polygala tenuifolia attenuates cognitive deficits by rescuing hippocampal neurogenesis in APP/PS1 transgenic mice

27-Hydroxycholesterol contributes to cognitive deficits in APP/PS1 transgenic mice through microbiota dysbiosis and intestinal barrier dysfunction

Pridopidine Promotes Synaptogenesis and Reduces Spatial Memory Deficits in the Alzheimer’s Disease APP/PS1 Mouse Model

Long-term inhibition of ODC1 in APP/PS1 mice rescues amyloid pathology and switches astrocytes from a reactive to active state

Urolithin A attenuates memory impairment and neuroinflammation in APP/PS1 mice

Influenza vaccination in early Alzheimer’s disease rescues amyloidosis and ameliorates cognitive deficits in APP/PS1 mice by inhibiting regulatory T cells

Inhibition of DYRK1A proteolysis modifies its kinase specificity and rescues Alzheimer phenotype in APP/PS1 mice

Pan-PPAR Modulation Effectively Protects APP/PS1 Mice from Amyloid Deposition and Cognitive Deficits

Immune senescence in aged APP/PS1 mice

Deletion of aquaporin-4 in APP/PS1 mice exacerbates brain Aβ accumulation and memory deficits

Rescue of cognitive deficits in APP/PS1 mice by accelerating the aggregation of β-amyloid peptide

Minor ginsenoside F1 improves memory in APP/PS1 mice

Active constituent of Polygala tenuifolia attenuates cognitive deficits by rescuing hippocampal neurogenesis in APP/PS1 transgenic mice

27-Hydroxycholesterol contributes to cognitive deficits in APP/PS1 transgenic mice through microbiota dysbiosis and intestinal barrier dysfunction

Pridopidine Promotes Synaptogenesis and Reduces Spatial Memory Deficits in the Alzheimer’s Disease APP/PS1 Mouse Model

Long-term inhibition of ODC1 in APP/PS1 mice rescues amyloid pathology and switches astrocytes from a reactive to active state

Urolithin A attenuates memory impairment and neuroinflammation in APP/PS1 mice

Influenza vaccination in early Alzheimer’s disease rescues amyloidosis and ameliorates cognitive deficits in APP/PS1 mice by inhibiting regulatory T cells

Inhibition of DYRK1A proteolysis modifies its kinase specificity and rescues Alzheimer phenotype in APP/PS1 mice

Pan-PPAR Modulation Effectively Protects APP/PS1 Mice from Amyloid Deposition and Cognitive Deficits

Immune senescence in aged APP/PS1 mice

Deletion of aquaporin-4 in APP/PS1 mice exacerbates brain Aβ accumulation and memory deficits

Rescue of cognitive deficits in APP/PS1 mice by accelerating the aggregation of β-amyloid peptide

Minor ginsenoside F1 improves memory in APP/PS1 mice

Active constituent of Polygala tenuifolia attenuates cognitive deficits by rescuing hippocampal neurogenesis in APP/PS1 transgenic mice

27-Hydroxycholesterol contributes to cognitive deficits in APP/PS1 transgenic mice through microbiota dysbiosis and intestinal barrier dysfunction

Pridopidine Promotes Synaptogenesis and Reduces Spatial Memory Deficits in the Alzheimer’s Disease APP/PS1 Mouse Model

Long-term inhibition of ODC1 in APP/PS1 mice rescues amyloid pathology and switches astrocytes from a reactive to active state

Urolithin A attenuates memory impairment and neuroinflammation in APP/PS1 mice

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