Repeated electroacupuncture treatment attenuated hyperalgesia through suppression of spinal glial activation in chronic neuropathic pain rats
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1472-6882
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
-
online resource.
- Erschienen in
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Repeated electroacupuncture treatment attenuated hyperalgesia through suppression of spinal glial activation in chronic neuropathic pain rats ; volume:18 ; number:1 ; day:21 ; month:2 ; year:2018 ; pages:1-11 ; date:12.2018
BMC complementary and alternative medicine ; 18, Heft 1 (21.2.2018), 1-11, 12.2018
- Urheber
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Wang, Jun-ying
- Beteiligte Personen und Organisationen
-
Gao, Yong-hui
Qiao, Li-na
Zhang, Jian-liang
Duan-mu, Cheng-Lin
Yan, Ya-xia
Chen, Shu-ping
Liu, Jun-ling
SpringerLink (Online service)
- DOI
-
10.1186/s12906-018-2134-8
- URN
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urn:nbn:de:1111-201804175789
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:48 MESZ
Datenpartner
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Beteiligte
- Wang, Jun-ying
- Gao, Yong-hui
- Qiao, Li-na
- Zhang, Jian-liang
- Duan-mu, Cheng-Lin
- Yan, Ya-xia
- Chen, Shu-ping
- Liu, Jun-ling
- SpringerLink (Online service)
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The persistent release of HMGB1 contributes to tactile hyperalgesia in a rodent model of neuropathic pain
Electroacupuncture remediates glial dysfunction and ameliorates neurodegeneration in the astrocytic α-synuclein mutant mouse model
Dextromethorphan attenuated the higher vulnerability to inflammatory thermal hyperalgesia caused by prenatal morphine exposure in rat offspring
Involvement of adenosine A1 receptor in electroacupuncture-mediated inhibition of astrocyte activation during neuropathic pain
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Blocking spinal CCR2 with AZ889 reversed hyperalgesia in a model of neuropathic pain
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The persistent release of HMGB1 contributes to tactile hyperalgesia in a rodent model of neuropathic pain
Electroacupuncture remediates glial dysfunction and ameliorates neurodegeneration in the astrocytic α-synuclein mutant mouse model
Dextromethorphan attenuated the higher vulnerability to inflammatory thermal hyperalgesia caused by prenatal morphine exposure in rat offspring
Involvement of adenosine A1 receptor in electroacupuncture-mediated inhibition of astrocyte activation during neuropathic pain
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