Overweight worsens apoptosis, neuroinflammation and blood-brain barrier damage after hypoxic ischemia in neonatal brain through JNK hyperactivation

to related objects

Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1742-2094

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Overweight worsens apoptosis, neuroinflammation and blood-brain barrier damage after hypoxic ischemia in neonatal brain through JNK hyperactivation ; volume:8 ; number:1 ; day:25 ; month:4 ; year:2011 ; pages:1-15 ; date:12.2011
Journal of neuroinflammation ; 8, Heft 1 (25.4.2011), 1-15, 12.2011

Creator: Tu, Yi-Fang

Contributor: Tsai, Yau-Sheng
Wang, Lan-Wan
Wu, Hsin-Chieh
Huang, Chao-Ching
Ho, Chien-Jung
SpringerLink (Online service)

DOI: 10.1186/1742-2094-8-40

URN: urn:nbn:de:1111-2016032329777

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:55 AM CEST

Data provider

This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.

Show original at data provider

Associated

Tu, Yi-Fang
Tsai, Yau-Sheng
Wang, Lan-Wan
Wu, Hsin-Chieh
Huang, Chao-Ching
Ho, Chien-Jung
SpringerLink (Online service)

Other Objects (12)

Neuro-inflammation, blood-brain barrier, seizures and autism

JNK signaling is the shared pathway linking neuroinflammation, blood–brain barrier disruption, and oligodendroglial apoptosis in the white matter injury of the immature brain

Hypoxic oligodendrocyte precursor cell-derived VEGFA is associated with blood–brain barrier impairment

TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain

Mfsd2a attenuated hypoxic-ischemic brain damage via protection of the blood–brain barrier in mfat-1 transgenic mice

Serum biomarkers of neuroinflammation and blood-brain barrier leakage in amyotrophic lateral sclerosis

Brain endothelial LRP1 maintains blood–brain barrier integrity

NKG2D knockdown improves hypoxic-ischemic brain damage by inhibiting neuroinflammation in neonatal mice

ASK1-K716R reduces neuroinflammation and white matter injury via preserving blood–brain barrier integrity after traumatic brain injury

Neuroinflammation and Brain Disease

Lipocalin 2 induces neuroinflammation and blood-brain barrier dysfunction through liver-brain axis in murine model of nonalcoholic steatohepatitis

Hochschulschrift

Function of MMP-2 and MMP-9 at the blood-brain barrier in neuroinflammation

Neuro-inflammation, blood-brain barrier, seizures and autism

JNK signaling is the shared pathway linking neuroinflammation, blood–brain barrier disruption, and oligodendroglial apoptosis in the white matter injury of the immature brain

Hypoxic oligodendrocyte precursor cell-derived VEGFA is associated with blood–brain barrier impairment

TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain

Mfsd2a attenuated hypoxic-ischemic brain damage via protection of the blood–brain barrier in mfat-1 transgenic mice

Serum biomarkers of neuroinflammation and blood-brain barrier leakage in amyotrophic lateral sclerosis

Brain endothelial LRP1 maintains blood–brain barrier integrity

NKG2D knockdown improves hypoxic-ischemic brain damage by inhibiting neuroinflammation in neonatal mice

ASK1-K716R reduces neuroinflammation and white matter injury via preserving blood–brain barrier integrity after traumatic brain injury

Neuroinflammation and Brain Disease

Lipocalin 2 induces neuroinflammation and blood-brain barrier dysfunction through liver-brain axis in murine model of nonalcoholic steatohepatitis

Hochschulschrift

Function of MMP-2 and MMP-9 at the blood-brain barrier in neuroinflammation

Neuro-inflammation, blood-brain barrier, seizures and autism

JNK signaling is the shared pathway linking neuroinflammation, blood–brain barrier disruption, and oligodendroglial apoptosis in the white matter injury of the immature brain

Hypoxic oligodendrocyte precursor cell-derived VEGFA is associated with blood–brain barrier impairment

TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain

Mfsd2a attenuated hypoxic-ischemic brain damage via protection of the blood–brain barrier in mfat-1 transgenic mice

Serum biomarkers of neuroinflammation and blood-brain barrier leakage in amyotrophic lateral sclerosis

Brain endothelial LRP1 maintains blood–brain barrier integrity

NKG2D knockdown improves hypoxic-ischemic brain damage by inhibiting neuroinflammation in neonatal mice

ASK1-K716R reduces neuroinflammation and white matter injury via preserving blood–brain barrier integrity after traumatic brain injury

Neuroinflammation and Brain Disease

Lipocalin 2 induces neuroinflammation and blood-brain barrier dysfunction through liver-brain axis in murine model of nonalcoholic steatohepatitis

Hochschulschrift

Function of MMP-2 and MMP-9 at the blood-brain barrier in neuroinflammation

Cultural heritage institutions wishing to register will find more information here.

Fields marked * need to be filled in.

Username*

Please enter your username

Email*

Please enter your email address

Please do not fill this field

First name

Last name

Password*

Please enter your password

Confirm password*

Please enter the same password

I have read the terms of use and the privacy policy for the collection of personal data and accept them. *

This field is required.

I would like to subscribe to the newsletter of the Deutsche Digitale Bibliothek. See newsletter subscription info.

Account created

Your "My DDB" account has been successfully created. Before you can log in to your account, you must click the confirmation link in the message we just sent to the email address you provided.

Overweight worsens apoptosis, neuroinflammation and blood-brain barrier damage after hypoxic ischemia in neonatal brain through JNK hyperactivation

Object Details

References and Relationships

Contributors, Places and Time

Further information

Data provider

Associated

Other Objects (12)

Neuro-inflammation, blood-brain barrier, seizures and autism

JNK signaling is the shared pathway linking neuroinflammation, blood–brain barrier disruption, and oligodendroglial apoptosis in the white matter injury of the immature brain

Hypoxic oligodendrocyte precursor cell-derived VEGFA is associated with blood–brain barrier impairment

TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain

Mfsd2a attenuated hypoxic-ischemic brain damage via protection of the blood–brain barrier in mfat-1 transgenic mice

Serum biomarkers of neuroinflammation and blood-brain barrier leakage in amyotrophic lateral sclerosis

Brain endothelial LRP1 maintains blood–brain barrier integrity

NKG2D knockdown improves hypoxic-ischemic brain damage by inhibiting neuroinflammation in neonatal mice

ASK1-K716R reduces neuroinflammation and white matter injury via preserving blood–brain barrier integrity after traumatic brain injury

Neuroinflammation and Brain Disease

Lipocalin 2 induces neuroinflammation and blood-brain barrier dysfunction through liver-brain axis in murine model of nonalcoholic steatohepatitis

Function of MMP-2 and MMP-9 at the blood-brain barrier in neuroinflammation

Neuro-inflammation, blood-brain barrier, seizures and autism

JNK signaling is the shared pathway linking neuroinflammation, blood–brain barrier disruption, and oligodendroglial apoptosis in the white matter injury of the immature brain

Hypoxic oligodendrocyte precursor cell-derived VEGFA is associated with blood–brain barrier impairment

TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain

Mfsd2a attenuated hypoxic-ischemic brain damage via protection of the blood–brain barrier in mfat-1 transgenic mice

Serum biomarkers of neuroinflammation and blood-brain barrier leakage in amyotrophic lateral sclerosis

Brain endothelial LRP1 maintains blood–brain barrier integrity

NKG2D knockdown improves hypoxic-ischemic brain damage by inhibiting neuroinflammation in neonatal mice

ASK1-K716R reduces neuroinflammation and white matter injury via preserving blood–brain barrier integrity after traumatic brain injury

Neuroinflammation and Brain Disease

Lipocalin 2 induces neuroinflammation and blood-brain barrier dysfunction through liver-brain axis in murine model of nonalcoholic steatohepatitis

Function of MMP-2 and MMP-9 at the blood-brain barrier in neuroinflammation

Neuro-inflammation, blood-brain barrier, seizures and autism

JNK signaling is the shared pathway linking neuroinflammation, blood–brain barrier disruption, and oligodendroglial apoptosis in the white matter injury of the immature brain

Hypoxic oligodendrocyte precursor cell-derived VEGFA is associated with blood–brain barrier impairment

TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain

Mfsd2a attenuated hypoxic-ischemic brain damage via protection of the blood–brain barrier in mfat-1 transgenic mice

Serum biomarkers of neuroinflammation and blood-brain barrier leakage in amyotrophic lateral sclerosis

Brain endothelial LRP1 maintains blood–brain barrier integrity

NKG2D knockdown improves hypoxic-ischemic brain damage by inhibiting neuroinflammation in neonatal mice

ASK1-K716R reduces neuroinflammation and white matter injury via preserving blood–brain barrier integrity after traumatic brain injury

Neuroinflammation and Brain Disease

Lipocalin 2 induces neuroinflammation and blood-brain barrier dysfunction through liver-brain axis in murine model of nonalcoholic steatohepatitis

Function of MMP-2 and MMP-9 at the blood-brain barrier in neuroinflammation

Related objects

Reset password