- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
1 Online-Ressource.
- Language
-
Englisch
- Bibliographic citation
-
Inhibition and degradation of NRAS with a pan-NRAS monobody ; volume:43 ; number:48 ; day:8 ; month:10 ; year:2024 ; pages:3489-3497 ; date:11.2024
Oncogene ; 43, Heft 48 (8.10.2024), 3489-3497, 11.2024
- Classification
-
Medizin, Gesundheit
- Creator
-
Whaby, Michael
Ketavarapu, Gayatri
Koide, Akiko
Mazzei, Megan
Mintoo, Mubashir
Glasser, Eliezra
Patel, Unnatiben
Nasarre, Cecile
Sale, Matthew J.
McCormick, Frank
Koide, Shohei
O’Bryan, John P.
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1038/s41388-024-03186-y
- URN
-
urn:nbn:de:101:1-2502082200550.666592530234
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:26 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Whaby, Michael
- Ketavarapu, Gayatri
- Koide, Akiko
- Mazzei, Megan
- Mintoo, Mubashir
- Glasser, Eliezra
- Patel, Unnatiben
- Nasarre, Cecile
- Sale, Matthew J.
- McCormick, Frank
- Koide, Shohei
- O’Bryan, John P.
- SpringerLink (Online service)
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Die Rolle onkogener NRAS-Mutationen in der kolorektalen Tumorgenese
Targeting retinoid receptors with dysregulated microNRAs in pancreatic duct adenocarcinoma
Heterogeneous distribution of BRAF/NRAS mutations among Italian patients with advanced melanoma
Missense mutation of NRAS is associated with malignant progression in neurocutaneous melanosis
Experimental treatment of NRAS-mutated neurocutaneous melanocytosis with MEK162, a MEK-inhibitor
Induction of N-Ras degradation by flunarizine-mediated autophagy
Combination of pan-RAF and MEK inhibitors in NRAS mutant melanoma
NRAS destines tumor cells to the lungs
MEK targeting in N-RAS mutated metastatic melanoma
NRAS und SMAD4 als Biomarker bei fortgeschrittenem Pankreaskarzinom
STAT3 – the switch of melanoma-associated NRAS mutations
Immune checkpoint inhibition in patients with NRAS mutated and NRAS wild type melanoma: a multicenter Dermatologic Cooperative Oncology Group study on 637 patients from the prospective skin cancer registry ADOREG
Die Rolle onkogener NRAS-Mutationen in der kolorektalen Tumorgenese
Targeting retinoid receptors with dysregulated microNRAs in pancreatic duct adenocarcinoma
Heterogeneous distribution of BRAF/NRAS mutations among Italian patients with advanced melanoma
Missense mutation of NRAS is associated with malignant progression in neurocutaneous melanosis
Experimental treatment of NRAS-mutated neurocutaneous melanocytosis with MEK162, a MEK-inhibitor
Induction of N-Ras degradation by flunarizine-mediated autophagy
Combination of pan-RAF and MEK inhibitors in NRAS mutant melanoma
NRAS destines tumor cells to the lungs
MEK targeting in N-RAS mutated metastatic melanoma
NRAS und SMAD4 als Biomarker bei fortgeschrittenem Pankreaskarzinom
STAT3 – the switch of melanoma-associated NRAS mutations
Immune checkpoint inhibition in patients with NRAS mutated and NRAS wild type melanoma: a multicenter Dermatologic Cooperative Oncology Group study on 637 patients from the prospective skin cancer registry ADOREG
Die Rolle onkogener NRAS-Mutationen in der kolorektalen Tumorgenese
Targeting retinoid receptors with dysregulated microNRAs in pancreatic duct adenocarcinoma
Heterogeneous distribution of BRAF/NRAS mutations among Italian patients with advanced melanoma
Missense mutation of NRAS is associated with malignant progression in neurocutaneous melanosis