Anti-gp41 antibodies inhibit infection and transcytosis of HIV-1 infectious molecular clones expressing transmitted/founder envelopes
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1742-4690
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Anti-gp41 antibodies inhibit infection and transcytosis of HIV-1 infectious molecular clones expressing transmitted/founder envelopes ; volume:9 ; number:2 ; day:13 ; month:9 ; year:2012 ; pages:1 ; date:9.2012
Retrovirology ; 9, Heft 2 (13.9.2012), 1, 9.2012
- Creator
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Jain, S.
- Contributor
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Ochsenbauer, C.
Kappes, JC
Rosenthal, KL
SpringerLink (Online service)
- DOI
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10.1186/1742-4690-9-S2-P195
- URN
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urn:nbn:de:1111-2016080727603
- Rights
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Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:59 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Jain, S.
- Ochsenbauer, C.
- Kappes, JC
- Rosenthal, KL
- SpringerLink (Online service)
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Conformational flexibility of HIV-1 envelope glycoproteins modulates transmitted/founder sensitivity to broadly neutralizing antibodies
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Unique inclusions of maximal C-clones in maximal clones
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Green envelopes
Tanztheater "Envelopes"
A deeper view of transmitted/founder viruses using 454 whole genome deep sequencing
Direct identification of HLA‐presented CD8 T cell epitopes from transmitted founder HIV‐1 variants
Conformational flexibility of HIV-1 envelope glycoproteins modulates transmitted/founder sensitivity to broadly neutralizing antibodies
Local clones
False discovery rate envelopes
Envelopes in Banach spaces
P19-14. Recombinant alphavirus replicon particles as a platform to evaluate immunogenicity of early transmitted clade C virus envelopes
The number and genetic relatedness of transmitted/founder virus impact clinical outcome in vaginal R5 SHIVSF162P3N infection
Unique inclusions of maximal C-clones in maximal clones