- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1756-6606
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Autophagic and endo-lysosomal dysfunction in neurodegenerative disease ; volume:12 ; number:1 ; day:29 ; month:11 ; year:2019 ; pages:1-21 ; date:12.2019
Molecular brain ; 12, Heft 1 (29.11.2019), 1-21, 12.2019
- Creator
-
Malik, Bilal R.
Maddison, Daniel C.
Smith, Gaynor A.
Peters, Owen M.
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1186/s13041-019-0504-x
- URN
-
urn:nbn:de:101:1-2020032423153579531532
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:46 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Malik, Bilal R.
- Maddison, Daniel C.
- Smith, Gaynor A.
- Peters, Owen M.
- SpringerLink (Online service)
Other Objects (12)
Lysosomal Ca2+ release-facilitated TFEB nuclear translocation alleviates ischemic brain injury by attenuating autophagic/lysosomal dysfunction in neurons
Lysosomal function in neurodegenerative diseases
Nanomechanical action opens endo-lysosomal compartments
N-ethylmaleimide-sensitive factor elicits a neuroprotection against ischemic neuronal injury by restoring autophagic/lysosomal dysfunction
Lysosomal dysfunction in proteinopathic neurodegenerative disorders: possible therapeutic roles of cAMP and zinc
Inhibition of endo-lysosomal function exacerbates vascular calcification
Pathways of integrins in the endo-lysosomal system
Triamterene induces autophagic degradation of lysosome by exacerbating lysosomal integrity
Indomethacin Disrupts Autophagic Flux by Inducing Lysosomal Dysfunction in Gastric Cancer Cells and Increases Their Sensitivity to Cytotoxic Drugs
Correction to: Pathways of integrins in the endo-lysosomal system
Neurodegenerative changes initiated by presynaptic dysfunction
Defects of Vps15 in skeletal muscles lead to autophagic vacuolar myopathy and lysosomal disease
Lysosomal Ca2+ release-facilitated TFEB nuclear translocation alleviates ischemic brain injury by attenuating autophagic/lysosomal dysfunction in neurons
Lysosomal function in neurodegenerative diseases
Nanomechanical action opens endo-lysosomal compartments
N-ethylmaleimide-sensitive factor elicits a neuroprotection against ischemic neuronal injury by restoring autophagic/lysosomal dysfunction
Lysosomal dysfunction in proteinopathic neurodegenerative disorders: possible therapeutic roles of cAMP and zinc
Inhibition of endo-lysosomal function exacerbates vascular calcification
Pathways of integrins in the endo-lysosomal system
Triamterene induces autophagic degradation of lysosome by exacerbating lysosomal integrity
Indomethacin Disrupts Autophagic Flux by Inducing Lysosomal Dysfunction in Gastric Cancer Cells and Increases Their Sensitivity to Cytotoxic Drugs
Correction to: Pathways of integrins in the endo-lysosomal system
Neurodegenerative changes initiated by presynaptic dysfunction
Defects of Vps15 in skeletal muscles lead to autophagic vacuolar myopathy and lysosomal disease
Lysosomal Ca2+ release-facilitated TFEB nuclear translocation alleviates ischemic brain injury by attenuating autophagic/lysosomal dysfunction in neurons
Lysosomal function in neurodegenerative diseases
Nanomechanical action opens endo-lysosomal compartments
N-ethylmaleimide-sensitive factor elicits a neuroprotection against ischemic neuronal injury by restoring autophagic/lysosomal dysfunction
Lysosomal dysfunction in proteinopathic neurodegenerative disorders: possible therapeutic roles of cAMP and zinc
Inhibition of endo-lysosomal function exacerbates vascular calcification
Pathways of integrins in the endo-lysosomal system
Triamterene induces autophagic degradation of lysosome by exacerbating lysosomal integrity
Indomethacin Disrupts Autophagic Flux by Inducing Lysosomal Dysfunction in Gastric Cancer Cells and Increases Their Sensitivity to Cytotoxic Drugs
Correction to: Pathways of integrins in the endo-lysosomal system
Neurodegenerative changes initiated by presynaptic dysfunction