Metabolic stress in cancer cells induces immune escape through a PI3K-dependent blockade of IFNγ receptor signaling
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2051-1426
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Metabolic stress in cancer cells induces immune escape through a PI3K-dependent blockade of IFNγ receptor signaling ; volume:7 ; number:1 ; day:13 ; month:6 ; year:2019 ; pages:1-12 ; date:12.2019
Journal for ImmunoTherapy of Cancer ; 7, Heft 1 (13.6.2019), 1-12, 12.2019
- Creator
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Marijt, Koen A.
Sluijter, Marjolein
Blijleven, Laura
Tolmeijer, Sofie H.
Scheeren, Ferenc A.
Burg, Sjoerd H. van der
Hall, Thorbald van
- Contributor
-
SpringerLink (Online service)
- DOI
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10.1186/s40425-019-0627-8
- URN
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urn:nbn:de:101:1-2019112601281004944278
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:49 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Marijt, Koen A.
- Sluijter, Marjolein
- Blijleven, Laura
- Tolmeijer, Sofie H.
- Scheeren, Ferenc A.
- Burg, Sjoerd H. van der
- Hall, Thorbald van
- SpringerLink (Online service)
Other Objects (12)
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PI3K induces B-cell development and regulates B cell identity
Absence of IFNγ expression induces neuronal degeneration in the spinal cord of adult mice
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Non-canonical deubiquitination of OTUB1 induces IFNγ-mediated cell cycle arrest via regulation of p27 stability
IRAK1-regulated IFN-γ signaling induces MDSC to facilitate immune evasion in FGFR1-driven hematological malignancies
Mismatch in epitope specificities between IFNγ inflamed and uninflamed conditions leads to escape from T lymphocyte killing in melanoma
Escape
ESCAPE
Interferon (IFNγ) induces cell cycle arrest in RCC cell lines
Roxarsone induces angiogenesis via PI3K/Akt signaling
MITF induces escape from innate immunity in melanoma
A neuropeptide, Substance-P, directly induces tissue-repairing M2 like macrophages by activating the PI3K/Akt/mTOR pathway even in the presence of IFNγ
PI3K induces B-cell development and regulates B cell identity
Absence of IFNγ expression induces neuronal degeneration in the spinal cord of adult mice
Author Correction: PI3K/AKT activation induces PTEN ubiquitination and destabilization accelerating tumourigenesis
Non-canonical deubiquitination of OTUB1 induces IFNγ-mediated cell cycle arrest via regulation of p27 stability
IRAK1-regulated IFN-γ signaling induces MDSC to facilitate immune evasion in FGFR1-driven hematological malignancies
Mismatch in epitope specificities between IFNγ inflamed and uninflamed conditions leads to escape from T lymphocyte killing in melanoma
Escape
ESCAPE
Interferon (IFNγ) induces cell cycle arrest in RCC cell lines
Roxarsone induces angiogenesis via PI3K/Akt signaling
MITF induces escape from innate immunity in melanoma
A neuropeptide, Substance-P, directly induces tissue-repairing M2 like macrophages by activating the PI3K/Akt/mTOR pathway even in the presence of IFNγ
PI3K induces B-cell development and regulates B cell identity
Absence of IFNγ expression induces neuronal degeneration in the spinal cord of adult mice
Author Correction: PI3K/AKT activation induces PTEN ubiquitination and destabilization accelerating tumourigenesis
Non-canonical deubiquitination of OTUB1 induces IFNγ-mediated cell cycle arrest via regulation of p27 stability
IRAK1-regulated IFN-γ signaling induces MDSC to facilitate immune evasion in FGFR1-driven hematological malignancies
Mismatch in epitope specificities between IFNγ inflamed and uninflamed conditions leads to escape from T lymphocyte killing in melanoma
Escape