The regenerative effect of stem cells on acetaminophen-induced hepatotoxicity in male albino rats
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2090-6226
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
-
online resource.
- Erschienen in
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The regenerative effect of stem cells on acetaminophen-induced hepatotoxicity in male albino rats ; volume:12 ; number:1 ; day:26 ; month:7 ; year:2022 ; pages:1-9 ; date:12.2022
Egyptian liver journal ; 12, Heft 1 (26.7.2022), 1-9, 12.2022
- Urheber
-
El-Araby, Hanaa Ahmed
Sobhy, Gihan Ahmed
Naeem, Suhaib Alsayed Mohammed
Alsayed, Aly Fahmy Mohamed
Zakaria, Haidy Mohammed
Khedr, Mohammed Ahmed
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1186/s43066-022-00206-y
- URN
-
urn:nbn:de:101:1-2022100719520624629949
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:20 MESZ
Datenpartner
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Beteiligte
- El-Araby, Hanaa Ahmed
- Sobhy, Gihan Ahmed
- Naeem, Suhaib Alsayed Mohammed
- Alsayed, Aly Fahmy Mohamed
- Zakaria, Haidy Mohammed
- Khedr, Mohammed Ahmed
- SpringerLink (Online service)
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High mobility group B1 impairs hepatocyte regeneration in acetaminophen hepatotoxicity
HMGB1 neutralization is associated with bacterial translocation during acetaminophen hepatotoxicity
Hepatoprotective effects of safranal on acetaminophen-induced hepatotoxicity in rats
Prolonged treatment with N-acetylcystine delays liver recovery from acetaminophen hepatotoxicity
Ecto-Nucleotide Triphosphate Diphosphohydrolase-2 (NTPDase2) Deletion Increases Acetaminophen-Induced Hepatotoxicity
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Mesenchymal stem cells protect against acetaminophen hepatotoxicity by secreting regenerative cytokine hepatocyte growth factor
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Sasa veitchii extracts suppress acetaminophen-induced hepatotoxicity in mice
The biochemistry of acetaminophen hepatotoxicity and rescue: a mathematical model
High mobility group B1 impairs hepatocyte regeneration in acetaminophen hepatotoxicity
HMGB1 neutralization is associated with bacterial translocation during acetaminophen hepatotoxicity
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Prolonged treatment with N-acetylcystine delays liver recovery from acetaminophen hepatotoxicity
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Bifidobacterium adolescentis-derived hypaphorine alleviates acetaminophen hepatotoxicity by promoting hepatic Cry1 expression