MIG‐6 is essential for promoting glucose metabolic reprogramming and tumor growth in triple‐negative breast cancer
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Erschienen in
-
MIG‐6 is essential for promoting glucose metabolic reprogramming and tumor growth in triple‐negative breast cancer ; year:2021
EMBO reports / European Molecular Biology Organization ; (2021)
- Urheber
-
He, Jiabei
Li, Chien‐Feng
Lee, Hong‐Jen
Shin, Dong‐Hui
Chern, Yi‐Jye
Pereira De Carvalho, Bruno
Chan, Chia‐Hsin
- DOI
-
10.15252/embr.202050781
- URN
-
urn:nbn:de:101:1-2021030510441490545944
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:27 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- He, Jiabei
- Li, Chien‐Feng
- Lee, Hong‐Jen
- Shin, Dong‐Hui
- Chern, Yi‐Jye
- Pereira De Carvalho, Bruno
- Chan, Chia‐Hsin
Ähnliche Objekte (12)
TGF-β induced reprogramming and drug resistance in triple-negative breast cells
Myc derived circRNA promotes triple-negative breast cancer progression via reprogramming fatty acid metabolism
Key regulator PNPLA8 drives phospholipid reprogramming induced proliferation and migration in triple-negative breast cancer
Metastatic Triple-Negative Breast Cancer
Transcriptome reprogramming through alternative splicing triggered by apigenin drives cell death in triple-negative breast cancer
Characterizing triple-negative breast cancer tumors
Triple-Negative Breast Cancer: Multimodality Appearance
TWEAK/Fn14 signalling driven super-enhancer reprogramming promotes pro-metastatic metabolic rewiring in triple-negative breast cancer
Overexpression of SLAP2 inhibits triple-negative breast cancer progression by promoting macrophage M1-type polarization
The chromatin architectural regulator SND1 mediates metastasis in triple-negative breast cancer by promoting CDH1 gene methylation
MicroRNA signatures in triple-negative breast cancer
Immune-related biomarkers in triple-negative breast cancer
TGF-β induced reprogramming and drug resistance in triple-negative breast cells
Myc derived circRNA promotes triple-negative breast cancer progression via reprogramming fatty acid metabolism
Key regulator PNPLA8 drives phospholipid reprogramming induced proliferation and migration in triple-negative breast cancer
Metastatic Triple-Negative Breast Cancer
Transcriptome reprogramming through alternative splicing triggered by apigenin drives cell death in triple-negative breast cancer
Characterizing triple-negative breast cancer tumors
Triple-Negative Breast Cancer: Multimodality Appearance
TWEAK/Fn14 signalling driven super-enhancer reprogramming promotes pro-metastatic metabolic rewiring in triple-negative breast cancer
Overexpression of SLAP2 inhibits triple-negative breast cancer progression by promoting macrophage M1-type polarization
The chromatin architectural regulator SND1 mediates metastasis in triple-negative breast cancer by promoting CDH1 gene methylation
MicroRNA signatures in triple-negative breast cancer
Immune-related biomarkers in triple-negative breast cancer
TGF-β induced reprogramming and drug resistance in triple-negative breast cells
Myc derived circRNA promotes triple-negative breast cancer progression via reprogramming fatty acid metabolism
Key regulator PNPLA8 drives phospholipid reprogramming induced proliferation and migration in triple-negative breast cancer
Metastatic Triple-Negative Breast Cancer
Transcriptome reprogramming through alternative splicing triggered by apigenin drives cell death in triple-negative breast cancer
Characterizing triple-negative breast cancer tumors
Triple-Negative Breast Cancer: Multimodality Appearance
TWEAK/Fn14 signalling driven super-enhancer reprogramming promotes pro-metastatic metabolic rewiring in triple-negative breast cancer
Overexpression of SLAP2 inhibits triple-negative breast cancer progression by promoting macrophage M1-type polarization
The chromatin architectural regulator SND1 mediates metastasis in triple-negative breast cancer by promoting CDH1 gene methylation
MicroRNA signatures in triple-negative breast cancer