Author Correction: Human cardiac stem cells rejuvenated by modulating autophagy with MHY-1685 enhance the therapeutic potential for cardiac repair
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2092-6413
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
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online resource.
- Erschienen in
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Author Correction: Human cardiac stem cells rejuvenated by modulating autophagy with MHY-1685 enhance the therapeutic potential for cardiac repair ; volume:54 ; number:7 ; day:26 ; month:7 ; year:2022 ; pages:1061-1063 ; date:7.2022
Experimental & molecular medicine ; 54, Heft 7 (26.7.2022), 1061-1063, 7.2022
- Urheber
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Park, Ji Hye
Kim, Hyeok
Moon, Hyung Ryong
Park, Bong-Woo
Park, Jae-Hyun
Sim, Woo-Sup
Kim, Jin-Ju
Lim, Hye Ji
Kim, Yeon-Ju
Ji, Seung Taek
Jang, Woong Bi
Rethineswaran, Vinoth Kumar
Van, Le Thi Hong
Giang, Ly Thanh Truong
Yun, Jisoo
Ha, Jong Seong
Ban, Kiwon
Chung, Hae Young
Baek, Sang Hong
Park, Hun-Jun
Kwon, Sang-Mo
- Beteiligte Personen und Organisationen
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SpringerLink (Online service)
- DOI
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10.1038/s12276-022-00807-y
- URN
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urn:nbn:de:101:1-2022102019283363816807
- Rechteinformation
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
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15.08.2025, 07:24 MESZ
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Beteiligte
- Park, Ji Hye
- Kim, Hyeok
- Moon, Hyung Ryong
- Park, Bong-Woo
- Park, Jae-Hyun
- Sim, Woo-Sup
- Kim, Jin-Ju
- Lim, Hye Ji
- Kim, Yeon-Ju
- Ji, Seung Taek
- Jang, Woong Bi
- Rethineswaran, Vinoth Kumar
- Van, Le Thi Hong
- Giang, Ly Thanh Truong
- Yun, Jisoo
- Ha, Jong Seong
- Ban, Kiwon
- Chung, Hae Young
- Baek, Sang Hong
- Park, Hun-Jun
- Kwon, Sang-Mo
- SpringerLink (Online service)
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Human cardiac stem cells rejuvenated by modulating autophagy with MHY-1685 enhance the therapeutic potential for cardiac repair
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Drug repurposing-based nanoplatform via modulating autophagy to enhance chemo-phototherapy against colorectal cancer
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Rhein attenuates angiotensin II-induced cardiac remodeling by modulating AMPK–FGF23 signaling
NADPH oxidase 2 mediates cardiac sympathetic denervation and myocyte autophagy, resulting in cardiac atrophy and dysfunction in doxorubicin-induced cardiomyopathy
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