Effect of CYP3A4*22, CYP3A5*3, and CYP3A combined genotypes on tamoxifen metabolism
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1432-1041
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Effect of CYP3A4*22, CYP3A5*3, and CYP3A combined genotypes on tamoxifen metabolism ; day:28 ; month:8 ; year:2017 ; pages:1-10
European journal of clinical pharmacology ; (28.8.2017), 1-10
- Keyword
-
Biopharmazie
Pharmakodynamik
Pharmakokinetik
- Creator
-
Sanchez Spitman, A. B.
- Contributor
-
Moes, D. J. A. R.
Gelderblom, H.
Dezentje, V. O.
Swen, J.J
Guchelaar, H. J.
SpringerLink (Online service)
- DOI
-
10.1007/s00228-017-2323-2
- URN
-
urn:nbn:de:1111-2017110523850
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:28 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Sanchez Spitman, A. B.
- Moes, D. J. A. R.
- Gelderblom, H.
- Dezentje, V. O.
- Swen, J.J
- Guchelaar, H. J.
- SpringerLink (Online service)
Other Objects (12)
Effect of CYP2C19 genotypes on tamoxifen metabolism and early-breast cancer relapse
CYP3A4 drug metabolism considerations in pediatric pharmacotherapy
Inflammation down-regulates CYP3A4-catalysed drug metabolism in hemodialysis patients
CYP2C8 rs11572080 and CYP3A4 rs2740574 risk genotypes in paclitaxel-treated premenopausal breast cancer patients
A novel method for predicting ligand regioselectivity to metabolism by the CYP3A4 enzyme
CYP3A4-mediated metabolism of artemisinin to 10β-hydroxyartemisinin with comparable anti-malarial potency
Predicting nicotine metabolism across ancestries using genotypes
CYP3A4 and CYP3A5genotyping by Pyrosequencing
Drug–Drug Interaction Studies of Esmethadone (REL-1017) Involving CYP3A4- and CYP2D6-Mediated Metabolism
Tamoxifen
Bacterial diet modulates tamoxifen-induced death via host fatty acid metabolism
Combined histone deacetylase inhibition and tamoxifen induces apoptosis in tamoxifen-resistant breast cancer models, by reversing Bcl-2 overexpression
Effect of CYP2C19 genotypes on tamoxifen metabolism and early-breast cancer relapse
CYP3A4 drug metabolism considerations in pediatric pharmacotherapy
Inflammation down-regulates CYP3A4-catalysed drug metabolism in hemodialysis patients
CYP2C8 rs11572080 and CYP3A4 rs2740574 risk genotypes in paclitaxel-treated premenopausal breast cancer patients
A novel method for predicting ligand regioselectivity to metabolism by the CYP3A4 enzyme
CYP3A4-mediated metabolism of artemisinin to 10β-hydroxyartemisinin with comparable anti-malarial potency
Predicting nicotine metabolism across ancestries using genotypes
CYP3A4 and CYP3A5genotyping by Pyrosequencing
Drug–Drug Interaction Studies of Esmethadone (REL-1017) Involving CYP3A4- and CYP2D6-Mediated Metabolism
Tamoxifen
Bacterial diet modulates tamoxifen-induced death via host fatty acid metabolism
Combined histone deacetylase inhibition and tamoxifen induces apoptosis in tamoxifen-resistant breast cancer models, by reversing Bcl-2 overexpression
Effect of CYP2C19 genotypes on tamoxifen metabolism and early-breast cancer relapse
CYP3A4 drug metabolism considerations in pediatric pharmacotherapy
Inflammation down-regulates CYP3A4-catalysed drug metabolism in hemodialysis patients
CYP2C8 rs11572080 and CYP3A4 rs2740574 risk genotypes in paclitaxel-treated premenopausal breast cancer patients
A novel method for predicting ligand regioselectivity to metabolism by the CYP3A4 enzyme
CYP3A4-mediated metabolism of artemisinin to 10β-hydroxyartemisinin with comparable anti-malarial potency
Predicting nicotine metabolism across ancestries using genotypes
CYP3A4 and CYP3A5genotyping by Pyrosequencing
Drug–Drug Interaction Studies of Esmethadone (REL-1017) Involving CYP3A4- and CYP2D6-Mediated Metabolism
Tamoxifen
Bacterial diet modulates tamoxifen-induced death via host fatty acid metabolism