Short-chain fatty acids and intestinal inflammation in multiple sclerosis: modulation of female susceptibility by microbial products?

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2038-3274

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Short-chain fatty acids and intestinal inflammation in multiple sclerosis: modulation of female susceptibility by microbial products? ; volume:12 ; number:1 ; day:7 ; month:4 ; year:2021 ; pages:1-8 ; date:12.2021
Autoimmunity highlights ; 12, Heft 1 (7.4.2021), 1-8, 12.2021

Creator: Becker, Anouck
Abuazab, Mosab
Schwiertz, Andreas
Walter, Silke
Faßbender, Klaus C.
Fousse, Mathias
Unger, Marcus M.

Contributor: SpringerLink (Online service)

DOI: 10.1186/s13317-021-00149-1

URN: urn:nbn:de:101:1-2021052909024604208949

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:50 AM CEST

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Becker, Anouck
Abuazab, Mosab
Schwiertz, Andreas
Walter, Silke
Faßbender, Klaus C.
Fousse, Mathias
Unger, Marcus M.
SpringerLink (Online service)

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Short-chain fatty acids and intestinal inflammation in multiple sclerosis: modulation of female susceptibility by microbial products?

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Other Objects (12)

Dietary fatty acids and susceptibility to multiple sclerosis

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Short-chain fatty acids in cancer pathogenesis

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Supplementing the early diet of broilers with soy protein concentrate can improve intestinal development and enhance short-chain fatty acid-producing microbes and short-chain fatty acids, especially butyric acid

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Supplementing the early diet of broilers with soy protein concentrate can improve intestinal development and enhance short-chain fatty acid-producing microbes and short-chain fatty acids, especially butyric acid

Faecal bacterial and short-chain fatty acids signature in hypercholesterolemia

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Gastric infusion of short-chain fatty acids can improve intestinal barrier function in weaned piglets

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Microbiota-derived short-chain fatty acids promote Th1 cell IL-10 production to maintain intestinal homeostasis

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